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arsenic trioxide and Genome Instability

arsenic trioxide has been researched along with Genome Instability in 4 studies

Research

Studies (4)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	4 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Hernández, A; Marcos, R; Peremartí, J; Ramos, F	1
Agar, G; Arslan, E; Aydin, M; Erturk, FA; Sigmaz, B; Taspinar, MS; Yagci, S	1
DiRisio, AC; Field, MS; Kamynina, E; Lachenauer, ER; Liebenthal, RP; Stover, PJ	1
Brenneman, MA; Chen, JD; Denissova, NG; Hakhverdyan, Z; Nasello, C; Yeung, PL	1

Other Studies

4 other study(ies) available for arsenic trioxide and Genome Instability

Article	Year
Arsenic exposure disrupts the normal function of the FA/BRCA repair pathway. Toxicological sciences : an official journal of the Society of Toxicology, 2014, Volume: 142, Issue:1 Topics: Arsenic Trioxide; Arsenicals; Cell Cycle; Cell Line; Cell Proliferation; Cell Survival; DNA Repair; Fanconi Anemia; Fanconi Anemia Complementation Group D2 Protein; Fibroblasts; Flow Cytometry; Genomic Instability; Humans; Micronucleus Tests; Oxides; Signal Transduction; Toxicity Tests, Acute	2014
Effects of As2O3 on DNA methylation, genomic instability, and LTR retrotransposon polymorphism in Zea mays. Environmental science and pollution research international, 2015, Volume: 22, Issue:23 Topics: Arsenic Trioxide; Arsenicals; DNA Methylation; Genomic Instability; Oxides; Polymorphism, Genetic; Retroelements; Seedlings; Terminal Repeat Sequences; Zea mays	2015
Arsenic trioxide targets MTHFD1 and SUMO-dependent nuclear de novo thymidylate biosynthesis. Proceedings of the National Academy of Sciences of the United States of America, 2017, 03-21, Volume: 114, Issue:12 Topics: Aminohydrolases; Animals; Arsenic Trioxide; Arsenicals; Cell Line; Cell Nucleus; Fibroblasts; Formate-Tetrahydrofolate Ligase; Genomic Instability; Glycine Hydroxymethyltransferase; Humans; Methylenetetrahydrofolate Dehydrogenase (NADP); Mice; Mice, Knockout; Multienzyme Complexes; Oxides; Proteolysis; Small Ubiquitin-Related Modifier Proteins; Sumoylation; Thymidine Monophosphate; Thymidylate Synthase; Uracil	2017
Promyelocytic leukemia nuclear bodies support a late step in DNA double-strand break repair by homologous recombination. Journal of cellular biochemistry, 2012, Volume: 113, Issue:5 Topics: Arsenic Trioxide; Arsenicals; Cell Nucleus; DNA Breaks, Double-Stranded; DNA End-Joining Repair; DNA, Neoplasm; Genomic Instability; Histones; Humans; Leukemia, Promyelocytic, Acute; Nuclear Proteins; Oncogene Proteins, Fusion; Oxides; Phosphorylation; Promyelocytic Leukemia Protein; Rad51 Recombinase; Recombinational DNA Repair; RNA Interference; Transcription Factors; Tretinoin; Tumor Suppressor Proteins	2012