arsenic trioxide has been researched along with Cancer of Gastrointestinal Tract in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (33.33) | 29.6817 |
| 2010's | 2 (66.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Jiang, M; Li, YH; Liu, LX; Ma, ZB; Xu, HY; Yang, YL | 1 |
| Bardsley, MR; Burgoyne, AM; Chan, JC; Chmielecki, J; Gao, F; Harismendy, O; Kendrick, ML; Lee, TE; Leonard, SY; Mao, J; Morosini, D; Ordog, T; Ross, JS; Shi, E; Sicklick, JK; Siena, M; Syed, SA; Tang, CM; Wang, K | 1 |
| Chen, YC; Huan, SY; Yang, CH | 1 |
1 review(s) available for arsenic trioxide and Cancer of Gastrointestinal Tract
| Article | Year |
|---|---|
Arsenic trioxide therapy for relapsed acute promyelocytic leukemia: an useful salvage therapy.
Topics: Acute Kidney Injury; Antineoplastic Agents; Apoptosis; Arrhythmias, Cardiac; Arsenic Trioxide; Arsenicals; Cell Differentiation; Chemical and Drug Induced Liver Injury; Drug Evaluation; Drug Screening Assays, Antitumor; Gastrointestinal Neoplasms; Humans; Leukemia, Promyelocytic, Acute; Life Tables; Medicine, Chinese Traditional; Mitochondria; Models, Biological; Neoplasm Proteins; Neoplastic Stem Cells; Neuromuscular Diseases; Oncogene Proteins, Fusion; Oxides; Remission Induction; Salvage Therapy; Survival Analysis; Treatment Outcome; Tumor Cells, Cultured; Urinary Bladder Neoplasms | 2000 |
2 other study(ies) available for arsenic trioxide and Cancer of Gastrointestinal Tract
| Article | Year |
|---|---|
Arsenic trioxide induces apoptosis of human gastrointestinal cancer cells.
Topics: Adenocarcinoma; Antineoplastic Agents; Apoptosis; Arsenic Trioxide; Arsenicals; Biomarkers, Tumor; Drug Administration Schedule; Gastrointestinal Neoplasms; Humans; Infusions, Intravenous; Oxides; Proto-Oncogene Proteins c-bcl-2; Time Factors; Treatment Outcome; Tumor Suppressor Protein p53 | 2014 |
Hedgehog pathway dysregulation contributes to the pathogenesis of human gastrointestinal stromal tumors via GLI-mediated activation of KIT expression.
Topics: Antineoplastic Agents; Arsenic Trioxide; Arsenicals; Binding Sites; Cell Line, Tumor; Cell Survival; Cilia; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Gastrointestinal Neoplasms; Gastrointestinal Stromal Tumors; Gene Expression Regulation, Neoplastic; Humans; Imatinib Mesylate; Interstitial Cells of Cajal; Mutation; Nerve Tissue Proteins; Nuclear Proteins; Oxides; Promoter Regions, Genetic; Proto-Oncogene Proteins c-kit; Receptor, Platelet-Derived Growth Factor alpha; RNA Interference; RNA, Messenger; Signal Transduction; Transfection; Zinc Finger Protein GLI1; Zinc Finger Protein Gli2; Zinc Finger Protein Gli3 | 2016 |