Page last updated: 2024-08-26

arginyl-glycyl-aspartic acid and Carcinogenesis

arginyl-glycyl-aspartic acid has been researched along with Carcinogenesis in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (66.67)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Attar-Schneider, O; Dabbah, M; Drucker, L; Jarchowsky-Dolberg, O; Lishner, M; Pasmanik-Chor, M; Tartakover Matalon, S1
Araki, N; Hide, T; Hirayama, M; Kobayashi, D; Komohara, Y; Kuratsu, J; Makino, K; Midorikawa, U; Mizuguchi, S; Nagai, M; Nagayama, M; Nakamura, H; Niibori-Nambu, A; Takeya, M; Takezaki, T; Tsubota, N1
Feng, MX; Fu, Y; Gu, JR; Li, J; Ma, MZ; Qin, WX; Wang, T; Xia, Q; Xue, F; Zhang, JJ; Zhang, ZG1

Other Studies

3 other study(ies) available for arginyl-glycyl-aspartic acid and Carcinogenesis

ArticleYear
Multiple myeloma BM-MSCs increase the tumorigenicity of MM cells via transfer of VLA4-enriched microvesicles.
    Carcinogenesis, 2020, 03-13, Volume: 41, Issue:1

    Topics: Aged; Aged, 80 and over; Bone Marrow; Carcinogenesis; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cell Separation; Cell-Derived Microparticles; Female; Flow Cytometry; Humans; Integrin alpha4beta1; Male; Mesenchymal Stem Cells; Middle Aged; Multiple Myeloma; Natalizumab; Neoplasm Staging; Oligopeptides; Primary Cell Culture; Proteomics; Tumor Microenvironment

2020
Glioma initiating cells form a differentiation niche via the induction of extracellular matrices and integrin αV.
    PloS one, 2013, Volume: 8, Issue:5

    Topics: Animals; Brain Neoplasms; Carcinogenesis; Cell Adhesion; Cell Differentiation; Cell Movement; Cell Survival; Dacarbazine; Disease Progression; Extracellular Matrix; Fibronectins; Gene Expression Profiling; Gene Expression Regulation, Neoplastic; Gene Ontology; Glioma; Humans; Integrin alphaV; Mice; Neoplasm Proteins; Neoplastic Stem Cells; Oligopeptides; Proteomics; Reproducibility of Results; RNA, Messenger; Spheroids, Cellular; Survival Analysis; Temozolomide

2013
Elevated autocrine EDIL3 protects hepatocellular carcinoma from anoikis through RGD-mediated integrin activation.
    Molecular cancer, 2014, Oct-01, Volume: 13

    Topics: Animals; Anoikis; Autocrine Communication; Calcium-Binding Proteins; Carcinogenesis; Carcinoma, Hepatocellular; Carrier Proteins; Cell Adhesion; Cell Adhesion Molecules; Cell Line, Tumor; Cell Proliferation; Female; Gene Expression Regulation, Neoplastic; Gene Knockdown Techniques; Humans; Integrin alphaV; Liver Neoplasms; Male; Mice, Nude; Middle Aged; Oligopeptides; Portal Vein; Prognosis; Reproducibility of Results; Signal Transduction; Thrombosis; Up-Regulation

2014