arginine and Oxaluria, Primary

arginine has been researched along with Oxaluria, Primary in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (33.33)18.2507
2000's0 (0.00)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Fillaus, JA; Plumb, TJ; Swee, ML1
Abramowicz, D; Acquaviva, C; Cochat, P; Durrbach, A; Fargue, S; Gagnadoux, MF; Girardin, E; Harambat, J; Janssen, F; Legendre, C; Liutkus, A; Macher, MA; Mourani, C; Nivet, H; Rolland, MO; Schott, AM; Tsimaratos, M1
Danpure, CJ; Leiper, JM; Oatey, PB1

Reviews

1 review(s) available for arginine and Oxaluria, Primary

ArticleYear
Nocturnal home hemodialysis for a patient with type 1 hyperoxaluria.
    American journal of kidney diseases : the official journal of the National Kidney Foundation, 2013, Volume: 62, Issue:6

    Topics: Adult; Amino Acid Substitution; Arginine; Circadian Rhythm; Combined Modality Therapy; DNA Mutational Analysis; Exons; Female; Glycine; Hemodialysis, Home; Humans; Hyperoxaluria, Primary; Kidney Failure, Chronic; Oxalates; Pyridoxine; Transaminases

2013

Other Studies

2 other study(ies) available for arginine and Oxaluria, Primary

ArticleYear
Genotype-phenotype correlation in primary hyperoxaluria type 1: the p.Gly170Arg AGXT mutation is associated with a better outcome.
    Kidney international, 2010, Volume: 77, Issue:5

    Topics: Amino Acid Substitution; Arginine; Child; Child, Preschool; Cohort Studies; Genotype; Heterozygote; Homozygote; Humans; Hyperoxaluria, Primary; Infant; Kidney Failure, Chronic; Mutation; Phenotype; Prognosis; Retrospective Studies; Transaminases

2010
Inhibition of alanine:glyoxylate aminotransferase 1 dimerization is a prerequisite for its peroxisome-to-mitochondrion mistargeting in primary hyperoxaluria type 1.
    The Journal of cell biology, 1996, Volume: 135, Issue:4

    Topics: Alanine Transaminase; Animals; Arginine; Biological Transport; COS Cells; Dimerization; Glycine; Humans; Hyperoxaluria, Primary; Leucine; Liver; Microbodies; Mitochondria; Point Mutation; Polymorphism, Genetic; Proline; Transaminases

1996