Page last updated: 2024-09-03

antibiotic g 418 and Hemophilia A

antibiotic g 418 has been researched along with Hemophilia A in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Feng, J; High, KA; Hill, KA; Margaritis, P; Scaringe, WA; Sommer, SS; Song, W; Tsai, B; Wang, J; Yang, C; Zhang, Y	1
Aghajan, M; Guo, S; Huang, L; Low, A; Monia, BP; Murray, SF; Quesenberry, T	1

Other Studies

2 other study(ies) available for antibiotic g 418 and Hemophilia A

Article	Year
A mouse model for nonsense mutation bypass therapy shows a dramatic multiday response to geneticin. Proceedings of the National Academy of Sciences of the United States of America, 2007, Sep-25, Volume: 104, Issue:39 Topics: Amebicides; Aminoglycosides; Animals; Antineoplastic Agents; Codon, Nonsense; Codon, Terminator; Disease Models, Animal; Gentamicins; Hemophilia A; Humans; Liver; Mice; Mice, Inbred C57BL; Models, Genetic; Protein Biosynthesis	2007
Targeting Translation Termination Machinery with Antisense Oligonucleotides for Diseases Caused by Nonsense Mutations. Nucleic acid therapeutics, 2019, Volume: 29, Issue:4 Topics: Animals; Codon, Nonsense; Disease Models, Animal; Factor IX; Gentamicins; Hemophilia A; Humans; Liver; Mice; Molecular Targeted Therapy; Oligonucleotides, Antisense; Peptide Chain Termination, Translational; Peptide Termination Factors; Protein Biosynthesis; RNA, Messenger	2019

Article

Year

A mouse model for nonsense mutation bypass therapy shows a dramatic multiday response to geneticin.

Proceedings of the National Academy of Sciences of the United States of America, 2007, Sep-25, Volume: 104, Issue:39

Topics: Amebicides; Aminoglycosides; Animals; Antineoplastic Agents; Codon, Nonsense; Codon, Terminator; Disease Models, Animal; Gentamicins; Hemophilia A; Humans; Liver; Mice; Mice, Inbred C57BL; Models, Genetic; Protein Biosynthesis

2007

Targeting Translation Termination Machinery with Antisense Oligonucleotides for Diseases Caused by Nonsense Mutations.

Nucleic acid therapeutics, 2019, Volume: 29, Issue:4

Topics: Animals; Codon, Nonsense; Disease Models, Animal; Factor IX; Gentamicins; Hemophilia A; Humans; Liver; Mice; Molecular Targeted Therapy; Oligonucleotides, Antisense; Peptide Chain Termination, Translational; Peptide Termination Factors; Protein Biosynthesis; RNA, Messenger

2019