aniracetam has been researched along with Stroke in 3 studies
Stroke: A group of pathological conditions characterized by sudden, non-convulsive loss of neurological function due to BRAIN ISCHEMIA or INTRACRANIAL HEMORRHAGES. Stroke is classified by the type of tissue NECROSIS, such as the anatomic location, vasculature involved, etiology, age of the affected individual, and hemorrhagic vs. non-hemorrhagic nature. (From Adams et al., Principles of Neurology, 6th ed, pp777-810)
| Excerpt | Relevance | Reference |
|---|---|---|
| "The effects of aniracetam, a cognition enhancer, on extracellular levels of glutamate (Glu), gamma-aminobutyric acid (GABA) and nitric oxide metabolites (NOx) were examined in the prefrontal cortex (PFC) and the basolateral amygdala (AMG) in stroke-prone spontaneously hypertensive rats (SHRSP) using in vivo microdialysis." | 7.71 | Aniracetam enhances glutamatergic transmission in the prefrontal cortex of stroke-prone spontaneously hypertensive rats. ( Matsumoto, M; Nakamura, K; Ohashi, S; Saito, H; Togashi, H; Ueno, K; Yoshioka, M, 2002) |
| "The effects of aniracetam on extracellular levels of dopamine (DA), serotonin (5-HT) and their metabolites were examined in five brain regions in freely moving stroke-prone spontaneously hypertensive rats (SHRSP) using in vivo microdialysis." | 3.71 | Site-specific activation of dopamine and serotonin transmission by aniracetam in the mesocorticolimbic pathway of rats. ( Koshikawa, N; Nakamura, K; Shirane, M, 2001) |
| "The effects of aniracetam, a cognition enhancer, on extracellular levels of glutamate (Glu), gamma-aminobutyric acid (GABA) and nitric oxide metabolites (NOx) were examined in the prefrontal cortex (PFC) and the basolateral amygdala (AMG) in stroke-prone spontaneously hypertensive rats (SHRSP) using in vivo microdialysis." | 3.71 | Aniracetam enhances glutamatergic transmission in the prefrontal cortex of stroke-prone spontaneously hypertensive rats. ( Matsumoto, M; Nakamura, K; Ohashi, S; Saito, H; Togashi, H; Ueno, K; Yoshioka, M, 2002) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (66.67) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 1 (33.33) | 2.80 |
| Authors | Studies |
|---|---|
| Sharma, H | 1 |
| Reeta, KH | 1 |
| Sharma, U | 1 |
| Suri, V | 1 |
| Singh, S | 1 |
| Nakamura, K | 2 |
| Shirane, M | 1 |
| Koshikawa, N | 1 |
| Togashi, H | 1 |
| Matsumoto, M | 1 |
| Ueno, K | 1 |
| Ohashi, S | 1 |
| Saito, H | 1 |
| Yoshioka, M | 1 |
3 other studies available for aniracetam and Stroke
| Article | Year |
|---|---|
AMPA receptor modulation through sequential treatment with perampanel and aniracetam mitigates post-stroke damage in experimental model of ischemic stroke.
Topics: alpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid; Animals; Anti-Inflammatory Agents; Infarct | 2023 |
Site-specific activation of dopamine and serotonin transmission by aniracetam in the mesocorticolimbic pathway of rats.
Topics: Amygdala; Animals; Behavior, Animal; Dopamine; Hippocampus; Male; Microdialysis; Motor Activity; Neu | 2001 |
Aniracetam enhances glutamatergic transmission in the prefrontal cortex of stroke-prone spontaneously hypertensive rats.
Topics: Amino Acids; Amygdala; Animals; Cerebral Infarction; Extracellular Space; gamma-Aminobutyric Acid; G | 2002 |