angiotensin ii, des-phe(8)- has been researched along with Pulmonary Disease, Chronic Obstructive in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 1 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| Heng, CKM; Liao, W; Mei, D; Tan, WSD; Wong, WSF | 1 |
| Fu, X; Li, Y; Shi, C; Song, Y; Zhang, Y; Zhao, L | 1 |
2 other study(ies) available for angiotensin ii, des-phe(8)- and Pulmonary Disease, Chronic Obstructive
| Article | Year |
|---|---|
Activation of angiotensin II type-2 receptor protects against cigarette smoke-induced COPD.
Topics: Airway Remodeling; Angiotensin I; Angiotensin II; Animals; Anti-Inflammatory Agents; Antioxidants; Cytokines; Disease Models, Animal; Female; Imidazoles; Inflammation Mediators; Lung; Macrophages, Alveolar; Mice, Inbred BALB C; Neutrophils; Oxidative Stress; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Pulmonary Disease, Chronic Obstructive; Pulmonary Emphysema; Receptor, Angiotensin, Type 2; Receptors, G-Protein-Coupled; Renin-Angiotensin System; Signal Transduction; Smoke; Sulfonamides; Thiophenes; Tobacco Products | 2020 |
Angiotensin-(1-7)-mediated Mas1 receptor/NF-κB-p65 signaling is involved in a cigarette smoke-induced chronic obstructive pulmonary disease mouse model.
Topics: Angiotensin I; Animals; Bronchoalveolar Lavage Fluid; Chemokine CXCL1; Disease Models, Animal; Enzyme-Linked Immunosorbent Assay; Injections, Subcutaneous; Interleukin-6; Lung; Male; Mice; Mice, Inbred C57BL; NF-KappaB Inhibitor alpha; Nicotiana; Peptide Fragments; Phosphorylation; Proto-Oncogene Mas; Proto-Oncogene Proteins; Pulmonary Disease, Chronic Obstructive; Pulmonary Fibrosis; Receptors, G-Protein-Coupled; Signal Transduction; Smoke; Transcription Factor RelA; Tumor Necrosis Factor-alpha | 2018 |