angiotensin ii, des-phe(8)- has been researched along with Disease Exacerbation in 14 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 4 (28.57) | 29.6817 |
2010's | 8 (57.14) | 24.3611 |
2020's | 2 (14.29) | 2.80 |
Authors | Studies |
---|---|
Aazami, H; Kamali, M; Khoshmirsafa, M; Mansouri, D; Mohsenzadegan, M; Pornour, M; Seif, F | 1 |
Arfsten, H; Bartko, PE; Domenig, O; Goliasch, G; Hengstenberg, C; Hülsmann, M; Mascherbauer, J; Pavo, N; Poglitsch, M; Prausmüller, S; Spinka, G; Strunk, G; Uyanik-Ünal, K; Wurm, R; Zuckermann, A | 1 |
Coletta, RD; de Oliveira, CE; Hinsley, EE; Hunt, S; Lambert, DW | 1 |
Amundson, E; Kashani, N; Kelland, EE; Lee, S; Levy, AM; Lund, BT; Rodgers, KE; Stone, R | 1 |
Guo, T; Hao, P; Kong, J; Li, D; Meng, X; Yang, J; Zhang, C; Zhang, K; Zhang, M; Zhang, Y | 1 |
Červenka, L; Husková, Z; Melenovský, V; Nishiyama, A; Sadowski, J; Škaroupková, P | 1 |
Gao, Q; Jiang, T; Lu, H; Tan, L; Tian, YY; Yu, JT; Zhang, YD; Zhao, HD; Zhou, JS; Zhu, XC | 1 |
Bricca, G; Kobeissy, F; Marcelo, P; Nasser, R; Nehme, A; Zibara, K | 1 |
Bader, M; Bürgelová, M; Cervenka, L; Dvorák, P; Kramer, HJ; Malý, J; Opocenský, M; Thumová, M; Vanourková, Z; Zelízko, M | 1 |
Casley, D; Gaspari, TA; Tesanovic, S; Vinh, A; Widdop, RE | 1 |
Alenina, N; Bader, M; Bürgelová, M; Cervenka, L; Husková, Z; Kramer, HJ; Malý, J; Mrázová, I; Netuka, I; Opočenský, M; Rakušan, D; Santos, RA; Skaroupková, P; Vaněčková, I; Vaňourková, Z | 1 |
Gamliel-Lazarovich, A; Kaplan, M; Keidar, S | 1 |
Diniz, JS; Pereira, RM; Pinheiro, SV; Santos, RA; Simões e Silva, AC | 1 |
Deng, BP; Dong, B; Dong, QL; Feng, JB; Li, SY; Liu, B; Liu, CX; Pan, CM; Song, HD; Yang, YP; Yu, QT; Zhang, C; Zhang, MX; Zhang, Y; Zhao, YX; Zhu, L | 1 |
2 review(s) available for angiotensin ii, des-phe(8)- and Disease Exacerbation
Article | Year |
---|---|
JAK Inhibition as a New Treatment Strategy for Patients with COVID-19.
Topics: Angiotensin I; Angiotensin-Converting Enzyme 2; Angiotensin-Converting Enzyme Inhibitors; Antiviral Agents; Azetidines; Betacoronavirus; Coronavirus Infections; COVID-19; Disease Progression; Gene Expression Regulation; Host-Pathogen Interactions; Humans; Janus Kinases; Methotrexate; Molecular Targeted Therapy; Pandemics; Peptide Fragments; Peptidyl-Dipeptidase A; Pneumonia, Viral; Purines; Pyrazoles; Receptor, Angiotensin, Type 1; SARS-CoV-2; Signal Transduction; STAT Transcription Factors; Sulfonamides | 2020 |
ACE2 of the heart: From angiotensin I to angiotensin (1-7).
Topics: Angiotensin I; Angiotensin-Converting Enzyme 2; Angiotensin-Converting Enzyme Inhibitors; Animals; Antihypertensive Agents; Disease Progression; Heart Failure; Humans; Myocardium; Peptide Fragments; Peptidyl-Dipeptidase A; Receptor, Angiotensin, Type 1; Ventricular Remodeling | 2007 |
12 other study(ies) available for angiotensin ii, des-phe(8)- and Disease Exacerbation
Article | Year |
---|---|
Myocardial Angiotensin Metabolism in End-Stage Heart Failure.
Topics: Angiotensin I; Angiotensin II; Angiotensin Receptor Antagonists; Angiotensin-Converting Enzyme Inhibitors; Disease Progression; Female; Heart Failure; Heart Transplantation; Humans; Male; Mass Spectrometry; Middle Aged; Myocardium; Peptide Fragments; Renin-Angiotensin System; Stroke Volume | 2021 |
Angiotensin 1-7 inhibits angiotensin II-stimulated head and neck cancer progression.
Topics: Angiotensin I; Angiotensin II; Cell Culture Techniques; Cell Movement; Disease Progression; Fibroblasts; Head and Neck Neoplasms; Humans; Immunoblotting; Peptide Fragments; Polymerase Chain Reaction; Renin-Angiotensin System; Signal Transduction; Transfection; Tumor Cells, Cultured | 2017 |
Reduced disease severity following therapeutic treatment with angiotensin 1-7 in a mouse model of multiple sclerosis.
Topics: Angiotensin I; Animals; Cell Proliferation; Disease Progression; Dose-Response Relationship, Drug; Encephalomyelitis, Autoimmune, Experimental; Male; Mice; Neuroprotection; Neuroprotective Agents; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Receptors, G-Protein-Coupled; Severity of Illness Index; Treatment Outcome | 2019 |
Angiotensin(1-7) attenuates the progression of streptozotocin-induced diabetic renal injury better than angiotensin receptor blockade.
Topics: Angiotensin I; Angiotensin II; Angiotensin II Type 1 Receptor Blockers; Animals; Cell Proliferation; Collagen Type IV; Diabetes Mellitus, Experimental; Diabetic Nephropathies; Disease Progression; Male; Mesangial Cells; Oxidative Stress; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Rats; Rats, Wistar; Receptors, G-Protein-Coupled; Signal Transduction; Smad3 Protein; Transforming Growth Factor beta1; Valsartan; Vascular Endothelial Growth Factor A | 2015 |
Inhibition of soluble epoxide hydrolase counteracts the development of renal dysfunction and progression of congestive heart failure in Ren-2 transgenic hypertensive rats with aorto-caval fistula.
Topics: Angiotensin I; Angiotensin II; Angiotensin-Converting Enzyme Inhibitors; Animals; Aorta; Blood Pressure; Cytochrome P-450 Enzyme System; Disease Progression; Enzyme Inhibitors; Epoxide Hydrolases; Fatty Acids, Monounsaturated; Female; Fistula; Heart Failure; Heart Rate; Hemodynamics; Kidney; Male; Peptide Fragments; Rats; Rats, Transgenic; Renin; Renin-Angiotensin System; Solubility; Time Factors; Vena Cava, Inferior | 2015 |
Angiotensin-(1-7) is Reduced and Inversely Correlates with Tau Hyperphosphorylation in Animal Models of Alzheimer's Disease.
Topics: Alzheimer Disease; Angiotensin I; Animals; Brain; Disease Models, Animal; Disease Progression; Male; Mice, Inbred C57BL; Mice, Transgenic; Peptide Fragments; Phosphorylation; tau Proteins | 2016 |
The kinetics of angiotensin-I metabolism in human carotid atheroma: An emerging role for angiotensin (1-7).
Topics: Angiotensin I; Angiotensin II; Angiotensin III; Carotid Artery Diseases; Chromatography, High Pressure Liquid; Disease Progression; Humans; Mass Spectrometry; Peptide Fragments; Plaque, Atherosclerotic; Time Factors | 2016 |
Impairment of the angiotensin-converting enzyme 2-angiotensin-(1-7)-Mas axis contributes to the acceleration of two-kidney, one-clip Goldblatt hypertension.
Topics: Angiotensin I; Angiotensin II; Animals; Blood Pressure; Cardiomegaly; Disease Models, Animal; Disease Progression; Hypertension, Renovascular; Infusion Pumps, Implantable; Peptide Fragments; Rats; Rats, Transgenic; Surgical Instruments; Telemetry; Vasodilator Agents | 2009 |
Vasoprotective and atheroprotective effects of angiotensin (1-7) in apolipoprotein E-deficient mice.
Topics: Acetylcholine; Angiotensin I; Angiotensin II; Angiotensin II Type 2 Receptor Blockers; Animals; Aortic Diseases; Apolipoproteins E; Atherosclerosis; Disease Models, Animal; Disease Progression; Dose-Response Relationship, Drug; Endothelium, Vascular; Imidazoles; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Nitric Oxide; Nitric Oxide Synthase Type III; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Pyridines; Receptor, Angiotensin, Type 2; Receptors, G-Protein-Coupled; Superoxides; Time Factors; Vasodilation; Vasodilator Agents | 2010 |
Knockout of angiotensin 1-7 receptor Mas worsens the course of two-kidney, one-clip Goldblatt hypertension: roles of nitric oxide deficiency and enhanced vascular responsiveness to angiotensin II.
Topics: Angiotensin I; Angiotensin II; Animals; Blood Pressure; Disease Progression; Hypertension, Renovascular; Male; Mice; Mice, Knockout; Nitric Oxide; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Receptors, G-Protein-Coupled; Surgical Instruments; Vasomotor System | 2010 |
Circulating renin Angiotensin system in childhood chronic renal failure: marked increase of Angiotensin-(1-7) in end-stage renal disease.
Topics: Adolescent; Angiotensin I; Angiotensin II; Angiotensins; Case-Control Studies; Child; Child, Preschool; Cross-Sectional Studies; Disease Progression; Female; Humans; Hypertension; Kidney Failure, Chronic; Male; Peptide Fragments; Peptidyl-Dipeptidase A; Renin; Renin-Angiotensin System | 2006 |
Overexpression of ACE2 enhances plaque stability in a rabbit model of atherosclerosis.
Topics: Adenoviridae; Angioplasty, Balloon; Angiotensin I; Angiotensin II; Angiotensin-Converting Enzyme 2; Animals; Aorta, Abdominal; Atherosclerosis; Cell Line; Cells, Cultured; Collagen; Diet, Atherogenic; Dietary Fats; Disease Models, Animal; Disease Progression; Genetic Vectors; Humans; Mice; Peptide Fragments; Peptidyl-Dipeptidase A; Proto-Oncogene Mas; Proto-Oncogene Proteins; Rabbits; Receptors, G-Protein-Coupled; Time Factors; Transduction, Genetic; Up-Regulation | 2008 |