angiotensin ii, des-phe(8)- has been researched along with Cancer of Nasopharynx in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Chien, CY; Chuang, HC; Hsu, YC; Lin, YT; Wang, HC; Yang, MY | 1 |
Chen, B; Chen, X; Du, H; Katovich, MJ; Li, A; Li, H; Li, J; Pei, N; Qi, Y; Sumners, C; Wan, R; Wei, W; Zhang, Y; Zheng, H | 1 |
2 other study(ies) available for angiotensin ii, des-phe(8)- and Cancer of Nasopharynx
Article | Year |
---|---|
Pre-treatment with angiotensin-(1-7) inhibits tumor growth via autophagy by downregulating PI3K/Akt/mTOR signaling in human nasopharyngeal carcinoma xenografts.
Topics: Angiotensin I; Animals; Autophagy; Cell Line, Tumor; Down-Regulation; Humans; Male; Mice, Nude; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; Peptide Fragments; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Signal Transduction; TOR Serine-Threonine Kinases; Xenograft Model Antitumor Assays | 2018 |
Angiotensin-(1-7) Decreases Cell Growth and Angiogenesis of Human Nasopharyngeal Carcinoma Xenografts.
Topics: Angiogenesis Inhibitors; Angiotensin I; Animals; Carcinoma; Cell Line, Tumor; Cell Movement; Cell Proliferation; Disease Models, Animal; Female; Humans; Hypoxia-Inducible Factor 1, alpha Subunit; MAP Kinase Signaling System; Membrane Proteins; Mice; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; Neovascularization, Pathologic; Peptide Fragments; Proto-Oncogene Mas; Receptors, Vascular Endothelial Growth Factor; Tumor Burden; Vascular Endothelial Growth Factor A; Xenograft Model Antitumor Assays | 2016 |