angiotensin ii, des-phe(8)- has been researched along with Becker Muscular Dystrophy in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Acuña, MJ; Bader, M; Brandan, E; Cabello-Verrugio, C; Cabrera, D; Muñoz-Canoves, P; Olguin, H; Pessina, P; Santos, RA; Vio, CP | 1 |
| Acuña, MJ; Brandan, E; Cabrera, D; Rebolledo, D; Riquelme, C; Santos, RA; Torrejón, J | 1 |
2 other study(ies) available for angiotensin ii, des-phe(8)- and Becker Muscular Dystrophy
| Article | Year |
|---|---|
Restoration of muscle strength in dystrophic muscle by angiotensin-1-7 through inhibition of TGF-β signalling.
Topics: Angiotensin I; Animals; Disease Models, Animal; Extracellular Matrix; Fibroblasts; Fibrosis; Humans; Male; Mice; Mice, Inbred mdx; Mice, Knockout; MicroRNAs; Muscle Strength; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Peptide Fragments; Receptors, Cell Surface; Signal Transduction; Transforming Growth Factor beta | 2014 |
ACE2 is augmented in dystrophic skeletal muscle and plays a role in decreasing associated fibrosis.
Topics: Angiotensin I; Angiotensin-Converting Enzyme 2; Animals; Cytoskeletal Proteins; Fibrosis; Humans; Inflammation; Mice; Mice, Inbred C57BL; Mice, Inbred mdx; Muscle Fibers, Skeletal; Muscular Dystrophy, Animal; Muscular Dystrophy, Duchenne; Peptide Fragments; Peptidyl-Dipeptidase A | 2014 |