angiotensin ii, des-phe(8)- has been researched along with Atrophy, Muscle in 9 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 8 (88.89) | 24.3611 |
| 2020's | 1 (11.11) | 2.80 |
| Authors | Studies |
|---|---|
| Abrigo, J; Aguirre, F; Cabello-Verrugio, C; Gonzalez, A; Gonzalez, F; Simon, F | 1 |
| Abrigo, J; Aravena, J; Araya-Durán, I; Cabello-Verrugio, C; González-Nilo, FD; Márquez-Miranda, V; Pacheco, N; Rivera, JC; Simon, F | 1 |
| Hall, SE; Winslow, MA | 1 |
| Abrigo, J; Brandan, E; Cabello-Verrugio, C; Cisternas, F; Meneses, C; Morales, MG; Simon, F; Vazquez, Y | 1 |
| Abrigo, J; Brandan, E; Cabello-Verrugio, C; Meneses, C; Morales, MG; Simon, F | 1 |
| Brandan, E; Cabello-Verrugio, C; Di Capua, G; Morales, MG; Olguín, H; Simon, F | 1 |
| Abrigo, J; Acuña, MJ; Bader, M; Brandan, E; Cabello-Verrugio, C; Cabrera, D; Morales, MG; Olguin, H; Santos, RA; Simon, F | 1 |
| Ábrigo, J; Cabello-Verrugio, C; Cabrera, D; Simon, F | 1 |
| Cabello-Verrugio, C; Garcia, D; Rivera, JC | 1 |
2 review(s) available for angiotensin ii, des-phe(8)- and Atrophy, Muscle
| Article | Year |
|---|---|
Muscle wasting: A review of exercise, classical and non-classical RAS axes.
Topics: Angiotensin I; Angiotensin II; Apoptosis; Exercise; Fibrosis; Humans; Mitochondria; Muscle, Skeletal; Muscular Atrophy; NADPH Oxidases; Peptide Fragments; Protein Biosynthesis; Reactive Oxygen Species; Renin-Angiotensin System; Ubiquitin-Protein Ligases | 2019 |
Skeletal muscle wasting: new role of nonclassical renin-angiotensin system.
Topics: Angiotensin I; Humans; Muscle, Skeletal; Muscular Atrophy; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Receptors, G-Protein-Coupled; Renin-Angiotensin System; Signal Transduction | 2017 |
7 other study(ies) available for angiotensin ii, des-phe(8)- and Atrophy, Muscle
| Article | Year |
|---|---|
Protective Effect of Angiotensin 1-7 on Sarcopenia Induced by Chronic Liver Disease in Mice.
Topics: Angiotensin I; Animals; Autophagy; Biomarkers; Chronic Disease; Fibrosis; Liver Diseases; Male; Mice; Mice, Inbred C57BL; Muscle Fibers, Skeletal; Muscle Proteins; Muscle Strength; Muscle, Skeletal; Muscular Atrophy; Peptide Fragments; Proteasome Endopeptidase Complex; Sarcopenia; SKP Cullin F-Box Protein Ligases; Tripartite Motif Proteins; Ubiquitin; Ubiquitin-Protein Ligases | 2020 |
The complex of PAMAM-OH dendrimer with Angiotensin (1-7) prevented the disuse-induced skeletal muscle atrophy in mice.
Topics: Angiotensin I; Animals; Dendrimers; Electrophoretic Mobility Shift Assay; Immobilization; Injections, Intraperitoneal; Male; Mice, Inbred C57BL; Molecular Dynamics Simulation; Muscle Proteins; Muscle, Skeletal; Muscular Atrophy; Muscular Disorders, Atrophic; Myosin Heavy Chains; Peptide Fragments; Peptides; SKP Cullin F-Box Protein Ligases; Static Electricity; Tripartite Motif Proteins; Ubiquitin-Protein Ligases | 2017 |
Angiotensin-(1-7) decreases skeletal muscle atrophy induced by angiotensin II through a Mas receptor-dependent mechanism.
Topics: Angiotensin I; Angiotensin II; Animals; Cell Line; Gene Expression Regulation; Male; Mice, Inbred C57BL; Muscle Fibers, Skeletal; Muscle Proteins; Muscle Strength; Muscle, Skeletal; Muscular Atrophy; Myosin Heavy Chains; Peptide Fragments; Phosphorylation; Proto-Oncogene Mas; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Receptors, G-Protein-Coupled; Signal Transduction; SKP Cullin F-Box Protein Ligases; Tripartite Motif Proteins; Ubiquitin-Protein Ligases | 2015 |
The angiotensin-(1-7)/Mas axis reduces myonuclear apoptosis during recovery from angiotensin II-induced skeletal muscle atrophy in mice.
Topics: Angiotensin I; Angiotensin II; Animals; Apoptosis; Disease Models, Animal; Immunoblotting; In Situ Nick-End Labeling; Male; Mice; Mice, Inbred C57BL; Muscle, Skeletal; Muscular Atrophy; Peptide Fragments; Polymerase Chain Reaction; Proto-Oncogene Mas; Proto-Oncogene Proteins; Receptors, G-Protein-Coupled | 2015 |
Endotoxin-induced skeletal muscle wasting is prevented by angiotensin-(1-7) through a p38 MAPK-dependent mechanism.
Topics: Analysis of Variance; Angiotensin I; Animals; Cells, Cultured; In Vitro Techniques; Lipopolysaccharides; Male; Mice; Mice, Inbred C57BL; Muscle Strength; Muscle, Skeletal; Muscular Atrophy; Myofibrils; p38 Mitogen-Activated Protein Kinases; Peptide Fragments; Random Allocation; Systemic Inflammatory Response Syndrome; Ubiquitin-Protein Ligases; Vasodilator Agents | 2015 |
Angiotensin-(1-7) attenuates disuse skeletal muscle atrophy in mice via its receptor, Mas.
Topics: Angiotensin I; Animals; Insulin-Like Growth Factor I; Isometric Contraction; Male; Mice; Mice, Inbred C57BL; Muscle Fibers, Skeletal; Muscle Proteins; Muscle Strength; Muscular Atrophy; Muscular Disorders, Atrophic; Myosin Heavy Chains; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Receptor, IGF Type 1; Receptors, G-Protein-Coupled; Signal Transduction; SKP Cullin F-Box Protein Ligases; Tripartite Motif Proteins; Ubiquitin-Protein Ligases | 2016 |
Angiotensin-(1-7) Prevents Skeletal Muscle Atrophy Induced by Transforming Growth Factor Type Beta (TGF-β) via Mas Receptor Activation.
Topics: Angiotensin I; Animals; Cell Line; Mice, Inbred C57BL; Muscle Fibers, Skeletal; Muscle Proteins; Muscle, Skeletal; Muscular Atrophy; Myosin Heavy Chains; Myosins; Peptide Fragments; Polyubiquitin; Proteasome Endopeptidase Complex; Proto-Oncogene Mas; Proto-Oncogene Proteins; Reactive Oxygen Species; Receptors, G-Protein-Coupled; Transforming Growth Factor beta; Tripartite Motif Proteins; Ubiquitin; Ubiquitin-Protein Ligases; Ubiquitination | 2016 |