Page last updated: 2024-09-03

angiotensin ii, des-phe(8)- and Aortic Diseases

angiotensin ii, des-phe(8)- has been researched along with Aortic Diseases in 4 studies

Research

Studies (4)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's4 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Dong, M; Gao, F; Hao, PP; Li, JJ; Liu, XL; Meng, X; Wang, XP; Yang, JM; Yang, XY; Zhang, C; Zhang, K; Zhang, MX; Zhang, Y; Zhao, XQ; Zhao, YX1
Dong, M; Guan, J; Meng, X; Niu, R; Sun, Y; Yang, J; Yang, X; Zhang, C; Zhang, Y1
Cao, Q; Gong, J; Liu, Y; Song, Y; Yu, M; Zhang, L1
Casley, D; Gaspari, TA; Tesanovic, S; Vinh, A; Widdop, RE1

Other Studies

4 other study(ies) available for angiotensin ii, des-phe(8)- and Aortic Diseases

ArticleYear
Angiotensin-(1-7) dose-dependently inhibits atherosclerotic lesion formation and enhances plaque stability by targeting vascular cells.
    Arteriosclerosis, thrombosis, and vascular biology, 2013, Volume: 33, Issue:8

    Topics: Angiotensin I; Animals; Aortic Diseases; Apolipoproteins E; Atherosclerosis; Blood Pressure; Body Weight; Cell Movement; Cell Proliferation; Collagen; Dose-Response Relationship, Drug; Lipids; Macrophages; Matrix Metalloproteinases; Mice; Mice, Knockout; Microfilament Proteins; Muscle Proteins; Muscle, Smooth, Vascular; Peptide Fragments; Receptor, Angiotensin, Type 2; RNA, Messenger; Vasodilator Agents

2013
Comparison of angiotensin-(1-7), losartan and their combination on atherosclerotic plaque formation in apolipoprotein E knockout mice.
    Atherosclerosis, 2015, Volume: 240, Issue:2

    Topics: Angiotensin I; Angiotensin II Type 1 Receptor Blockers; Animals; Aorta, Abdominal; Aortic Diseases; Apolipoproteins E; Atherosclerosis; Blood Pressure; Cell Line; Cell Movement; Cell Proliferation; Diet, High-Fat; Disease Models, Animal; Drug Therapy, Combination; Endothelium, Vascular; Humans; Lipids; Losartan; Macrophages; Male; Mice, Knockout; Muscle, Smooth, Vascular; Peptide Fragments; Plaque, Atherosclerotic; Renin-Angiotensin System; Superoxides; Time Factors; Vasodilation

2015
Soluble receptor for advanced glycation end products mitigates vascular dysfunction in spontaneously hypertensive rats.
    Molecular and cellular biochemistry, 2016, Volume: 419, Issue:1-2

    Topics: Angiotensin I; Angiotensin-Converting Enzyme 2; Animals; Aorta; Aortic Diseases; Gene Expression Regulation, Enzymologic; Glycation End Products, Advanced; Lactoylglutathione Lyase; NF-kappa B; Oxidative Stress; Peptide Fragments; Peptidyl-Dipeptidase A; PPAR gamma; Rats; Rats, Inbred SHR; Reactive Oxygen Species; Receptor for Advanced Glycation End Products; Vascular Remodeling

2016
Vasoprotective and atheroprotective effects of angiotensin (1-7) in apolipoprotein E-deficient mice.
    Arteriosclerosis, thrombosis, and vascular biology, 2010, Volume: 30, Issue:8

    Topics: Acetylcholine; Angiotensin I; Angiotensin II; Angiotensin II Type 2 Receptor Blockers; Animals; Aortic Diseases; Apolipoproteins E; Atherosclerosis; Disease Models, Animal; Disease Progression; Dose-Response Relationship, Drug; Endothelium, Vascular; Imidazoles; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Nitric Oxide; Nitric Oxide Synthase Type III; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Pyridines; Receptor, Angiotensin, Type 2; Receptors, G-Protein-Coupled; Superoxides; Time Factors; Vasodilation; Vasodilator Agents

2010