angiotensin ii, des-phe(8)- has been researched along with Airway Hyper-Responsiveness in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Akhtar, S; Babyson, RS; Benter, IF; El-Hashim, AZ; Ezeamuzie, CI; Khajah, MA; Renno, WM | 1 |
| Abreu, SC; Barcelos, LS; Caliari, MV; Campagnole-Santos, MJ; Magalhães, GS; Motta-Santos, D; Prata, LO; Rocco, PR; Rodrigues-Machado, MG; Santos, RA; Silva, AR | 1 |
2 other study(ies) available for angiotensin ii, des-phe(8)- and Airway Hyper-Responsiveness
| Article | Year |
|---|---|
Ang-(1-7)/ MAS1 receptor axis inhibits allergic airway inflammation via blockade of Src-mediated EGFR transactivation in a murine model of asthma.
Topics: Angiotensin I; Animals; Asthma; Blotting, Western; Bronchoalveolar Lavage Fluid; Chemotaxis, Leukocyte; Disease Models, Animal; ErbB Receptors; Fluorescent Antibody Technique; Lung; Male; Mice; Mice, Inbred BALB C; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Receptors, G-Protein-Coupled; Respiratory Hypersensitivity; Signal Transduction; src-Family Kinases | 2019 |
Angiotensin-(1-7) attenuates airway remodelling and hyperresponsiveness in a model of chronic allergic lung inflammation.
Topics: Airway Remodeling; Angiotensin I; Animals; Anti-Inflammatory Agents; Bronchial Hyperreactivity; Bronchoconstriction; Collagen; Cytokines; Disease Models, Animal; Hypertrophy, Right Ventricular; Immunoglobulin E; Inflammation Mediators; Lung; Male; Mice, Inbred BALB C; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Ovalbumin; Peptide Fragments; Phosphorylation; Pneumonia; Proto-Oncogene Mas; Proto-Oncogene Proteins; Receptors, G-Protein-Coupled; Respiratory Hypersensitivity; Signal Transduction | 2015 |