angiotensin ii, des-phe(8)- has been researched along with Acute Respiratory Distress Syndrome in 5 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (60.00) | 24.3611 |
2020's | 2 (40.00) | 2.80 |
Authors | Studies |
---|---|
Azuelos, I; Baglole, CJ; Eidelman, DH; Haidar, Z; Huang, MJ; Morganstein, T; Trivlidis, J | 1 |
Ciechanowicz, AK; Kucia, M; Lay, WX; Leszczak, C; Leszczak, S; Prado Paulino, J; Suchocki, E | 1 |
Bitker, L; Burrell, LM | 1 |
Chen, Q; Huang, Y; Liu, L; Pan, C; Qiu, H; Yang, Y | 1 |
Bos, AP; Florquin, S; Kamilic, J; Lutter, R; Moll, GN; Specht, PA; van der Loos, CM; van Goor, H; van Woensel, JB; Wösten-van Asperen, RM | 1 |
2 review(s) available for angiotensin ii, des-phe(8)- and Acute Respiratory Distress Syndrome
Article | Year |
---|---|
Involvement of the ACE2/Ang-(1-7)/MasR Axis in Pulmonary Fibrosis: Implications for COVID-19.
Topics: Angiotensin I; Angiotensin-Converting Enzyme 2; Cannabis; Cigarette Smoking; COVID-19; Fibrosis; Humans; Idiopathic Pulmonary Fibrosis; Inflammation; Lung; Pandemics; Peptide Fragments; Proto-Oncogene Mas; Respiration, Artificial; Respiratory Distress Syndrome; Respiratory Insufficiency; Risk Factors; SARS-CoV-2; Spike Glycoprotein, Coronavirus | 2021 |
Classic and Nonclassic Renin-Angiotensin Systems in the Critically Ill.
Topics: Acute Kidney Injury; Adult; Aged; Aged, 80 and over; Angiotensin I; Angiotensin II; Blood Pressure; Critical Illness; Female; Homeostasis; Humans; Male; Middle Aged; Peptide Fragments; Renin-Angiotensin System; Respiratory Distress Syndrome; Shock, Septic; Vasoconstrictor Agents | 2019 |
3 other study(ies) available for angiotensin ii, des-phe(8)- and Acute Respiratory Distress Syndrome
Article | Year |
---|---|
Angiotensin 1-7 Stimulates Proliferation of Lung Bronchoalveolar Progenitors-Implications for SARS-CoV-2 Infection.
Topics: Angiotensin I; Angiotensin-Converting Enzyme 2; Animals; Cell Proliferation; COVID-19; Inflammasomes; Lung; Mice; NLR Family, Pyrin Domain-Containing 3 Protein; Peptide Fragments; Peptidyl-Dipeptidase A; Renin-Angiotensin System; Respiratory Distress Syndrome; SARS-CoV-2 | 2022 |
Angiotensin-(1-7) attenuates lung fibrosis by way of Mas receptor in acute lung injury.
Topics: Acute Lung Injury; Angiotensin I; Angiotensin II; Angiotensin II Type 1 Receptor Blockers; Animals; Disease Models, Animal; Lipopolysaccharides; Losartan; Male; Mice; Mice, Inbred C57BL; Peptide Fragments; Proto-Oncogene Mas; Proto-Oncogene Proteins; Pulmonary Fibrosis; Receptors, G-Protein-Coupled; Respiratory Distress Syndrome; Vasodilator Agents | 2013 |
Acute respiratory distress syndrome leads to reduced ratio of ACE/ACE2 activities and is prevented by angiotensin-(1-7) or an angiotensin II receptor antagonist.
Topics: Angiotensin I; Angiotensin Receptor Antagonists; Angiotensin-Converting Enzyme 2; Animals; Bronchoalveolar Lavage Fluid; Disease Models, Animal; Intubation, Intratracheal; Lipopolysaccharides; Losartan; Lung; Macrophages, Alveolar; Male; Peptide Fragments; Peptidyl-Dipeptidase A; Rats; Rats, Sprague-Dawley; Respiratory Distress Syndrome | 2011 |