Page last updated: 2024-09-05

anacardic acid and Neoplasms

anacardic acid has been researched along with Neoplasms in 7 studies

Research

Studies (7)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (14.29)29.6817
2010's5 (71.43)24.3611
2020's1 (14.29)2.80

Authors

AuthorsStudies
da Silva, SR; Gunning, PT; Lukkarila, JL; Paiva, SL1
Hua, D; Wu, X1
Fernandez, HR; Lindén, SK1
Horibe, T; Kawakami, K; Okuno, Y; Torisawa, A1
Harsha Raj, M; Rössler, J; Salimath, BP; Yashaswini, B1
Ma, E; Park, WJ1
Chen, S; Jiang, X; Price, BD; Sun, Y1

Reviews

2 review(s) available for anacardic acid and Neoplasms

ArticleYear
Exploring a new frontier in cancer treatment: targeting the ubiquitin and ubiquitin-like activating enzymes.
    Journal of medicinal chemistry, 2013, Mar-28, Volume: 56, Issue:6

    Topics: Animals; Antineoplastic Agents; Humans; Molecular Targeted Therapy; Neoplasms; Ubiquitin; Ubiquitin-Activating Enzymes

2013
Small-molecule inhibitors targeting small ubiquitin-like modifier pathway for the treatment of cancers and other diseases.
    European journal of medicinal chemistry, 2022, Apr-05, Volume: 233

    Topics: Humans; Neoplasms; Protein Processing, Post-Translational; Small Ubiquitin-Related Modifier Proteins; Sumoylation; Ubiquitin

2022

Other Studies

5 other study(ies) available for anacardic acid and Neoplasms

ArticleYear
The aspirin metabolite salicylate inhibits lysine acetyltransferases and MUC1 induced epithelial to mesenchymal transition.
    Scientific reports, 2017, 07-17, Volume: 7, Issue:1

    Topics: Anacardic Acids; Cell Line, Tumor; Cell Proliferation; Cell Survival; Colorectal Neoplasms; Epithelial-Mesenchymal Transition; Gene Expression Regulation, Neoplastic; HT29 Cells; Humans; Lysine Acetyltransferases; Male; Mucin-1; Neoplasms; Phosphorylation; Prostatic Neoplasms; Proto-Oncogene Proteins c-akt; Salicylates

2017
Discovery of protein disulfide isomerase P5 inhibitors that reduce the secretion of MICA from cancer cells.
    Chembiochem : a European journal of chemical biology, 2014, Jul-21, Volume: 15, Issue:11

    Topics: Aminosalicylic Acids; Anacardic Acids; Benzenesulfonates; Cell Survival; Cells, Cultured; Dose-Response Relationship, Drug; Drug Discovery; Enzyme Inhibitors; HCT116 Cells; HeLa Cells; Histocompatibility Antigens Class I; Humans; Molecular Docking Simulation; Neoplasms; Protein Disulfide-Isomerases; Structure-Activity Relationship

2014
Combinatorial treatment with anacardic acid followed by TRAIL augments induction of apoptosis in TRAIL resistant cancer cells by the regulation of p53, MAPK and NFκβ pathways.
    Apoptosis : an international journal on programmed cell death, 2016, Volume: 21, Issue:5

    Topics: Anacardic Acids; Animals; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Apoptosis Regulatory Proteins; Caspase 3; Cell Line, Tumor; Drug Resistance, Neoplasm; Female; Humans; Liposomes; MAP Kinase Signaling System; Mice; Mitogen-Activated Protein Kinases; Neoplasms; NF-kappa B; Receptors, TNF-Related Apoptosis-Inducing Ligand; RNA, Messenger; Signal Transduction; TNF-Related Apoptosis-Inducing Ligand; Tumor Suppressor Protein p53; Up-Regulation

2016
Inhibition of PCAF histone acetyltransferase and cytotoxic effect of N-acylanthranilic acids.
    Archives of pharmacal research, 2012, Volume: 35, Issue:8

    Topics: Anacardic Acids; Antineoplastic Agents; Cell Line; Cell Line, Tumor; Enzyme Inhibitors; Humans; Neoplasms; ortho-Aminobenzoates; p300-CBP Transcription Factors; Structure-Activity Relationship

2012
Inhibition of histone acetyltransferase activity by anacardic acid sensitizes tumor cells to ionizing radiation.
    FEBS letters, 2006, Aug-07, Volume: 580, Issue:18

    Topics: Anacardic Acids; Ataxia Telangiectasia Mutated Proteins; Cell Cycle Proteins; Cell Line, Tumor; DNA-Activated Protein Kinase; DNA-Binding Proteins; Enzyme Inhibitors; HeLa Cells; Histone Acetyltransferases; Humans; Lysine Acetyltransferase 5; Neoplasms; Protein Serine-Threonine Kinases; Radiation-Sensitizing Agents; Radiation, Ionizing; Tumor Suppressor Proteins

2006