anacardic acid has been researched along with Muscular Dystrophy, Duchenne in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Barish, GD; Bass, J; Demonbreun, AR; Haldar, SM; Jiang, Z; Kuntz, NL; McNally, EM; Peek, CB; Quattrocelli, M; Zelikovich, AS | 1 |
Altucci, L; Berni, R; Capogrossi, MC; Castellano, S; Chimenti, C; Colussi, C; Frustaci, A; Gaetano, C; Macchi, E; Mai, A; Musso, E; Nebbioso, A; Rosati, J; Rossi, S; Sbardella, G; Spallotta, F; Stilli, D; Straino, S | 1 |
2 other study(ies) available for anacardic acid and Muscular Dystrophy, Duchenne
Article | Year |
---|---|
Pulsed glucocorticoids enhance dystrophic muscle performance through epigenetic-metabolic reprogramming.
Topics: Anacardic Acids; Animals; Biomarkers; Child; Cross-Sectional Studies; Disease Models, Animal; Drug Therapy, Combination; Epigenesis, Genetic; Epigenomics; Gene Expression Regulation; Glucocorticoids; Histone Acetyltransferases; Humans; Kruppel-Like Transcription Factors; Male; MEF2 Transcription Factors; Metabolomics; Mice; Mice, Inbred mdx; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Nutrients; Prednisone; Pulse Therapy, Drug | 2019 |
Nε-lysine acetylation determines dissociation from GAP junctions and lateralization of connexin 43 in normal and dystrophic heart.
Topics: Acetylation; Anacardic Acids; Animals; Cardiomyopathies; Connexin 43; Gap Junctions; Histone Acetyltransferases; Hydroxamic Acids; Immunoprecipitation; Lysine; Mice; Mice, Inbred mdx; Microscopy, Fluorescence; Muscular Dystrophy, Duchenne; Myocytes, Cardiac; p300-CBP Transcription Factors; Vorinostat | 2011 |