amphetamine has been researched along with Cerebral Infarction in 4 studies
Amphetamine: A powerful central nervous system stimulant and sympathomimetic. Amphetamine has multiple mechanisms of action including blocking uptake of adrenergics and dopamine, stimulation of release of monamines, and inhibiting monoamine oxidase. Amphetamine is also a drug of abuse and a psychotomimetic. The l- and the d,l-forms are included here. The l-form has less central nervous system activity but stronger cardiovascular effects. The d-form is DEXTROAMPHETAMINE.
1-phenylpropan-2-amine : A primary amine that is isopropylamine in which a hydrogen attached to one of the methyl groups has been replaced by a phenyl group.
amphetamine : A racemate comprising equimolar amounts of (R)-amphetamine (also known as levamphetamine or levoamphetamine) and (S)-amphetamine (also known as dexamfetamine or dextroamphetamine.
Cerebral Infarction: The formation of an area of NECROSIS in the CEREBRUM caused by an insufficiency of arterial or venous blood flow. Infarcts of the cerebrum are generally classified by hemisphere (i.e., left vs. right), lobe (e.g., frontal lobe infarction), arterial distribution (e.g., INFARCTION, ANTERIOR CEREBRAL ARTERY), and etiology (e.g., embolic infarction).
| Excerpt | Relevance | Reference |
|---|---|---|
| "The present studies were undertaken to examine 1) whether d-amphetamine sulfate administered to rats well after thrombotic infarction of the vibrissal cortical barrel-field within the primary somatosensory cortex affected the rate and completeness of behavioral recovery and 2) whether a dose-response relation exists between d-amphetamine sulfate dose and recovery of function." | 1.28 | Amphetamine promotes recovery from sensory-motor integration deficit after thrombotic infarction of the primary somatosensory rat cortex. ( Alonso, O; Dietrich, WD; Ginsberg, MD; Hurwitz, BE; McCabe, PM; Schneiderman, N; Watson, BD, 1991) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 4 (100.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Lambrecht, GL | 1 |
| Malbrain, ML | 1 |
| Chew, SL | 1 |
| Baeck, E | 1 |
| Verbraeken, H | 1 |
| Grabowski, M | 1 |
| Brundin, P | 1 |
| Johansson, BB | 1 |
| Roebroek, RM | 1 |
| Korten, JJ | 1 |
| Hurwitz, BE | 1 |
| Dietrich, WD | 1 |
| McCabe, PM | 1 |
| Alonso, O | 1 |
| Watson, BD | 1 |
| Ginsberg, MD | 1 |
| Schneiderman, N | 1 |
4 other studies available for amphetamine and Cerebral Infarction
| Article | Year |
|---|---|
Intranasal caffeine and amphetamine causing stroke.
Topics: Administration, Intranasal; Adult; Amphetamine; Brain; Caffeine; Cerebral Infarction; Humans; Magnet | 1993 |
Paw-reaching, sensorimotor, and rotational behavior after brain infarction in rats.
Topics: Amphetamine; Animals; Behavior, Animal; Cerebral Infarction; Disease Models, Animal; Forelimb; Male; | 1993 |
[Epileptic insults, cerebral infarct and rhabdomyolysis as complication of amphetamine use].
Topics: Amphetamine; Cerebral Infarction; Epilepsy; Humans; Male; Porphyria, Acute Intermittent; Rhabdomyoly | 1996 |
Amphetamine promotes recovery from sensory-motor integration deficit after thrombotic infarction of the primary somatosensory rat cortex.
Topics: Amphetamine; Animals; Cerebral Infarction; Learning; Male; Motor Activity; Nervous System Diseases; | 1991 |