Page last updated: 2024-10-25

amphetamine and Ascorbic Acid Deficiency

amphetamine has been researched along with Ascorbic Acid Deficiency in 1 studies

Amphetamine: A powerful central nervous system stimulant and sympathomimetic. Amphetamine has multiple mechanisms of action including blocking uptake of adrenergics and dopamine, stimulation of release of monamines, and inhibiting monoamine oxidase. Amphetamine is also a drug of abuse and a psychotomimetic. The l- and the d,l-forms are included here. The l-form has less central nervous system activity but stronger cardiovascular effects. The d-form is DEXTROAMPHETAMINE.
1-phenylpropan-2-amine : A primary amine that is isopropylamine in which a hydrogen attached to one of the methyl groups has been replaced by a phenyl group.
amphetamine : A racemate comprising equimolar amounts of (R)-amphetamine (also known as levamphetamine or levoamphetamine) and (S)-amphetamine (also known as dexamfetamine or dextroamphetamine.

Ascorbic Acid Deficiency: A condition due to a dietary deficiency of ascorbic acid (vitamin C), characterized by malaise, lethargy, and weakness. As the disease progresses, joints, muscles, and subcutaneous tissues may become the sites of hemorrhage. Ascorbic acid deficiency frequently develops into SCURVY in young children fed unsupplemented cow's milk exclusively during their first year. It develops also commonly in chronic alcoholism. (Cecil Textbook of Medicine, 19th ed, p1177)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19901 (100.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Matsuda, LA1
Schmidt, CJ1
Gibb, JW1
Hanson, GR1

Other Studies

1 other study available for amphetamine and Ascorbic Acid Deficiency

ArticleYear
Effects of methamphetamine on central monoaminergic systems in normal and ascorbic acid-deficient guinea pigs.
    Biochemical pharmacology, 1988, Sep-15, Volume: 37, Issue:18

    Topics: Amphetamine; Animals; Ascorbic Acid Deficiency; Brain; Corpus Striatum; Female; Guinea Pigs; Hydroxy

1988