ampelopsin has been researched along with Acute Liver Injury, Drug-Induced in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (25.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (50.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Basnet, P; Hase, K; Kadota, S; Namba, T; Ohsugi, M; Xiong, Q | 1 |
Awad, EM; El-Sheikh, AAK; El-Tahawy, NFG; Matouk, AI; Waz, S | 1 |
Dong, S; Hu, L; Ji, J; Wang, H | 1 |
Chen, TM; Dai, D; Hu, LP; Lan, Q; Liu, B; Liu, J; Xie, J; Zhang, QY; Zhang, WD; Zhu, RZ | 1 |
4 other study(ies) available for ampelopsin and Acute Liver Injury, Drug-Induced
Article | Year |
---|---|
Hepatoprotective effect of Hovenia dulcis THUNB. on experimental liver injuries induced by carbon tetrachloride or D-galactosamine/lipopolysaccharide.
Topics: Animals; Carbon Tetrachloride Poisoning; Cells, Cultured; Chemical and Drug Induced Liver Injury; Drugs, Chinese Herbal; Flavonoids; Galactosamine; Lipopolysaccharides; Liver; Male; Plant Extracts; Rats; Rats, Sprague-Dawley | 1997 |
Dihydromyricetin alleviates methotrexate-induced hepatotoxicity via suppressing the TLR4/NF-κB pathway and NLRP3 inflammasome/caspase 1 axis.
Topics: Animals; Anti-Inflammatory Agents; Antioxidants; Caspase 1; Chemical and Drug Induced Liver Injury; Cytokines; Flavonols; Glutathione; Inflammasomes; Interleukin-18; Interleukin-1beta; Malondialdehyde; Methotrexate; NF-kappa B; NLR Family, Pyrin Domain-Containing 3 Protein; Nucleotides; Rats; Toll-Like Receptor 4 | 2022 |
Dihydromyricetin alleviates acetaminophen-induced liver injury via the regulation of transformation, lipid homeostasis, cell death and regeneration.
Topics: Acetaminophen; Animals; Cell Death; Chemical and Drug Induced Liver Injury; Drugs, Chinese Herbal; Flavonols; Glutathione; Hepatocytes; Homeostasis; Lipid Metabolism; Lipids; Liver; Liver Regeneration; Male; Medicine, Chinese Traditional; Mice; Mice, Inbred C57BL; Signal Transduction | 2019 |
Dihydromyricetin alleviates carbon tetrachloride-induced acute liver injury via JNK-dependent mechanism in mice.
Topics: Animals; Anti-Inflammatory Agents; Biomarkers; Carbon Tetrachloride; Caspase Inhibitors; Cell Proliferation; Chemical and Drug Induced Liver Injury; Cytochromes c; Disease Models, Animal; Flavonols; Inflammation Mediators; JNK Mitogen-Activated Protein Kinases; Liver; Liver Failure, Acute; Liver Regeneration; Male; Mice, Inbred C57BL; Mitochondria, Liver; Protein Kinase Inhibitors; Signal Transduction; Time Factors; Tumor Necrosis Factor-alpha | 2015 |