ammonium hydroxide has been researched along with Status Epilepticus in 15 studies
azane : Saturated acyclic nitrogen hydrides having the general formula NnHn+2.
Status Epilepticus: A prolonged seizure or seizures repeated frequently enough to prevent recovery between episodes occurring over a period of 20-30 minutes. The most common subtype is generalized tonic-clonic status epilepticus, a potentially fatal condition associated with neuronal injury and respiratory and metabolic dysfunction. Nonconvulsive forms include petit mal status and complex partial status, which may manifest as behavioral disturbances. Simple partial status epilepticus consists of persistent motor, sensory, or autonomic seizures that do not impair cognition (see also EPILEPSIA PARTIALIS CONTINUA). Subclinical status epilepticus generally refers to seizures occurring in an unresponsive or comatose individual in the absence of overt signs of seizure activity. (From N Engl J Med 1998 Apr 2;338(14):970-6; Neurologia 1997 Dec;12 Suppl 6:25-30)
| Excerpt | Relevance | Reference |
|---|---|---|
| "Hyperammonemia is a common side effect of valproic acid (VPA) and can occur after generalized seizures, but the clinical significance is unclear." | 8.02 | Hyperammonemia in Patients With Status Epilepticus Treated With or Without Valproic Acid. ( Britton, JW; Hocker, SE; Smith, KM; Toledano, M, 2021) |
| "To evaluate the efficacy and tolerability of intravenous (IV) levetiracetam in refractory status epilepticus of migrating partial seizures in infancy (MPSI)." | 7.75 | Intravenous levetiracetam terminates refractory status epilepticus in two patients with migrating partial seizures in infancy. ( Balestri, M; Bianchi, R; Cilio, MR; Di Capua, M; Giovannini, S; Onofri, A; Vigevano, F, 2009) |
| "Hyperammonemia is a common side effect of valproic acid (VPA) and can occur after generalized seizures, but the clinical significance is unclear." | 4.02 | Hyperammonemia in Patients With Status Epilepticus Treated With or Without Valproic Acid. ( Britton, JW; Hocker, SE; Smith, KM; Toledano, M, 2021) |
| "To evaluate the efficacy and tolerability of intravenous (IV) levetiracetam in refractory status epilepticus of migrating partial seizures in infancy (MPSI)." | 3.75 | Intravenous levetiracetam terminates refractory status epilepticus in two patients with migrating partial seizures in infancy. ( Balestri, M; Bianchi, R; Cilio, MR; Di Capua, M; Giovannini, S; Onofri, A; Vigevano, F, 2009) |
| "Reversible neurotoxic symptoms were observed in three adult patients with absence status epilepticus on lamotrigine (LTG) therapy after administration of an IV bolus followed by oral valproic acid (VPA)." | 3.72 | Neurotoxicity following addition of intravenous valproate to lamotrigine therapy. ( Burneo, JG; Faught, E; Knowlton, RC; Kuzniecky, RI; Lawn, N; Limdi, N; Mendez, M; Prasad, A; Welty, TE, 2003) |
| "We observed a young patient with slight mental retardation, suffering from drug-resistant tonic-clonic seizures, who presented a status epilepticus (SE), in two separate periods when valproic acid (VPA) was added to the phenobarbital (PB) already being used in the patient's therapy." | 3.70 | Valproate-induced epileptic tonic status. ( Balducci, A; Capocchi, G; Cecconi, M; Pelli, MA; Picchiarelli, A; Silvestrelli, G; Zampolini, M, 1998) |
| "VPA is widely used for the treatment of generalized epilepsy." | 2.61 | [Valproic acid-induced hyperammonemic encephalopathy in a patient receiving valproic acid monotherapy]. ( Araki, M; Mukai, T; Naka, H; Shishido, T; Tokinobu, H; Yamada, H, 2019) |
| "We found no significant differences in seizure frequency, duration, severity, or administered KA doses before SE between the groups." | 1.56 | The effects of ammonia stimulation on kainate-induced status epilepticus and anterior piriform cortex electrophysiology. ( Bayat, A; Jones, A; Joshi, S; Koubeissi, MZ; Xiao, X, 2020) |
| "Primary hyperammonemic encephalopathy due to urea cycle disorders (UCD) typically manifests with episodic unresponsiveness and this clinical entity is not often included in the differential diagnosis of presumed non-convulsive status epilepticus (NCSE)." | 1.48 | Encephalopathy mimicking non-convulsive status Epilepticus. ( Al-Busaidi, A; Al-Busaidi, M; Al-Murshedi, F; Nandhagopal, R, 2018) |
| " Pharmacokinetic parameters are necessary to predict the optimum therapeutic level after administration." | 1.37 | Pharmacokinetics and clinical application of intravenous valproate in Thai epileptic children. ( Bhudhisawadi, K; Chulavatnatol, S; Kaojareon, S; Vaewpanich, J; Visudtibhan, A, 2011) |
| "The status epilepticus was resolved after antiepileptic therapy (phenytoin sodium) and treatment for hepatic encephalopathy (Branched chain amino acids)." | 1.33 | Hepatic encephalopathy with status epileptics: a case report. ( Hamagami, H; Ichinose, M; Kida, Y; Tanaka, H; Tsuji, T; Ueda, H, 2006) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (13.33) | 18.7374 |
| 1990's | 3 (20.00) | 18.2507 |
| 2000's | 5 (33.33) | 29.6817 |
| 2010's | 3 (20.00) | 24.3611 |
| 2020's | 2 (13.33) | 2.80 |
| Authors | Studies |
|---|---|
| Joshi, S | 1 |
| Bayat, A | 1 |
| Jones, A | 1 |
| Xiao, X | 1 |
| Koubeissi, MZ | 1 |
| Smith, KM | 1 |
| Britton, JW | 1 |
| Hocker, SE | 1 |
| Toledano, M | 1 |
| Nandhagopal, R | 1 |
| Al-Murshedi, F | 1 |
| Al-Busaidi, M | 1 |
| Al-Busaidi, A | 1 |
| Yamada, H | 1 |
| Shishido, T | 1 |
| Mukai, T | 1 |
| Araki, M | 1 |
| Naka, H | 1 |
| Tokinobu, H | 1 |
| Cilio, MR | 1 |
| Bianchi, R | 1 |
| Balestri, M | 1 |
| Onofri, A | 1 |
| Giovannini, S | 1 |
| Di Capua, M | 1 |
| Vigevano, F | 1 |
| Visudtibhan, A | 1 |
| Bhudhisawadi, K | 1 |
| Vaewpanich, J | 1 |
| Chulavatnatol, S | 1 |
| Kaojareon, S | 1 |
| Burneo, JG | 1 |
| Limdi, N | 1 |
| Kuzniecky, RI | 1 |
| Knowlton, RC | 1 |
| Mendez, M | 1 |
| Lawn, N | 1 |
| Faught, E | 1 |
| Welty, TE | 1 |
| Prasad, A | 1 |
| Tanaka, H | 1 |
| Ueda, H | 1 |
| Kida, Y | 1 |
| Hamagami, H | 1 |
| Tsuji, T | 1 |
| Ichinose, M | 1 |
| Velioğlu, SK | 1 |
| Gazioğlu, S | 1 |
| Tremolizzo, L | 1 |
| Galbussera, A | 1 |
| Frigo, M | 1 |
| Apale, P | 1 |
| Capra, M | 1 |
| Appollonio, I | 1 |
| Ferrarese, C | 1 |
| Sugimoto, T | 1 |
| Nishida, N | 1 |
| Yasuhara, A | 1 |
| Ono, A | 1 |
| Sakane, Y | 1 |
| Matsumura, T | 1 |
| Jagoda, A | 1 |
| Lichtenstein, GR | 1 |
| Kaiser, LR | 1 |
| Tuchman, M | 1 |
| Palevsky, HI | 1 |
| Kotloff, RM | 1 |
| O'Brien, CB | 1 |
| Furth, EE | 1 |
| Raps, EC | 1 |
| Berry, GT | 1 |
| Capocchi, G | 1 |
| Balducci, A | 1 |
| Cecconi, M | 1 |
| Pelli, MA | 1 |
| Picchiarelli, A | 1 |
| Silvestrelli, G | 1 |
| Zampolini, M | 1 |
| Kai, Y | 1 |
| Taku, K | 1 |
| Ohtani, Y | 1 |
| Miike, T | 1 |
| Miyazaki, T | 1 |
| Haraguchi, Y | 1 |
| Hattori, R | 1 |
1 review available for ammonium hydroxide and Status Epilepticus
| Article | Year |
|---|---|
[Valproic acid-induced hyperammonemic encephalopathy in a patient receiving valproic acid monotherapy].
Topics: Aged; Ammonia; Anticonvulsants; Biomarkers; Cardiomyopathies; Carnitine; Consciousness Disorders; Ep | 2019 |
14 other studies available for ammonium hydroxide and Status Epilepticus
| Article | Year |
|---|---|
The effects of ammonia stimulation on kainate-induced status epilepticus and anterior piriform cortex electrophysiology.
Topics: Ammonia; Animals; Electroencephalography; Kainic Acid; Male; Piriform Cortex; Rats; Rats, Sprague-Da | 2020 |
Hyperammonemia in Patients With Status Epilepticus Treated With or Without Valproic Acid.
Topics: Adult; Ammonia; Anticonvulsants; Humans; Hyperammonemia; Status Epilepticus; Valproic Acid | 2021 |
Encephalopathy mimicking non-convulsive status Epilepticus.
Topics: Adult; Ammonia; Brain Diseases; Diagnosis, Differential; Electroencephalography; Female; Humans; Mal | 2018 |
Intravenous levetiracetam terminates refractory status epilepticus in two patients with migrating partial seizures in infancy.
Topics: Ammonia; Anticonvulsants; Blood Cell Count; Creatine; Drug Administration Schedule; Electroencephalo | 2009 |
Pharmacokinetics and clinical application of intravenous valproate in Thai epileptic children.
Topics: Adolescent; Ammonia; Anticonvulsants; Child; Child, Preschool; Cross-Sectional Studies; Epilepsy; Fe | 2011 |
Neurotoxicity following addition of intravenous valproate to lamotrigine therapy.
Topics: Administration, Oral; Adult; Ammonia; Anticonvulsants; Confusion; Drug Interactions; Drug Therapy, C | 2003 |
Hepatic encephalopathy with status epileptics: a case report.
Topics: Amino Acids, Branched-Chain; Ammonia; Brain; Electroencephalography; Hepatic Encephalopathy; Humans; | 2006 |
Non-convulsive status epilepticus secondary to valproic acid-induced hyperammonemic encephalopathy.
Topics: Adult; Ammonia; Anticonvulsants; Brain; Electroencephalography; Hepatic Encephalopathy; Humans; Hype | 2007 |
Rapidly cycling encephalopathy from an almost forgotten entity.
Topics: Aged, 80 and over; Ammonia; Brain Diseases, Metabolic; Colon, Sigmoid; Consciousness Disorders; Diso | 2008 |
Reye-like syndrome associated with valproic acid.
Topics: Adolescent; Ammonia; Biopsy; Female; Humans; Liver; Liver Function Tests; Reye Syndrome; Status Epil | 1983 |
Altered mental status and nonconvulsive status epilepticus.
Topics: Ammonia; Epilepsy, Complex Partial; Humans; Mental Processes; Status Epilepticus | 1994 |
Fatal hyperammonemia following orthotopic lung transplantation.
Topics: Ammonia; Fatal Outcome; Female; Humans; Liver; Lung; Lung Transplantation; Metabolic Diseases; Micro | 1997 |
Valproate-induced epileptic tonic status.
Topics: Adolescent; Ammonia; Anticonvulsants; Drug Therapy, Combination; Electroencephalography; Epilepsy, T | 1998 |
[A case of ornithine transcarbamylase deficiency associated with a minor epileptic status].
Topics: Ammonia; Child; Electroencephalography; Female; Humans; Ornithine Carbamoyltransferase Deficiency Di | 1987 |