Page last updated: 2024-08-22

ammonium chloride and Reperfusion Injury

ammonium chloride has been researched along with Reperfusion Injury in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
da Silva, MT; de Brito, TS; de Menezes, DB; dos Santos, AA; Freire, RS; Libório, AB; Magalhães, PA; Martins, AM; Vale, ML1
Chen, NH; Chu, SF; Gao, Y; Song, XY; Wang, ZZ; Xia, CY; Yang, PF; Zhang, S; Zuo, W1
Arreola, J; Kaba, NK; Kim, M; Knauf, PA; Law, FY; Lefort, CT; Martel-Gallegos, G; Schultz, J; Waugh, RE1

Other Studies

3 other study(ies) available for ammonium chloride and Reperfusion Injury

ArticleYear
Metabolic acidosis aggravates experimental acute kidney injury.
    Life sciences, 2016, Feb-01, Volume: 146

    Topics: Acidosis; Acute Kidney Injury; Ammonium Chloride; Animals; Bicarbonates; Heme Oxygenase (Decyclizing); Hydrogen-Ion Concentration; Kidney Function Tests; Male; NF-kappa B; Nitric Oxide Synthase Type III; Rats; Rats, Wistar; Renal Artery; Reperfusion Injury; Water-Electrolyte Imbalance

2016
Autophagic flux regulates microglial phenotype according to the time of oxygen-glucose deprivation/reperfusion.
    International immunopharmacology, 2016, Volume: 39

    Topics: Ammonium Chloride; Animals; Autophagy; Brain Ischemia; Cell Line; Cyclic AMP Response Element-Binding Protein; Mice; Microglia; Microtubule-Associated Proteins; NF-kappa B; Nitric Oxide Synthase Type II; Phenotype; Reperfusion Injury; Transcription Factor TFIIH; Transcription Factors; Tumor Necrosis Factor-alpha

2016
Inhibition of Na+/H+ exchanger enhances low pH-induced L-selectin shedding and beta2-integrin surface expression in human neutrophils.
    American journal of physiology. Cell physiology, 2008, Volume: 295, Issue:5

    Topics: Amiloride; Ammonium Chloride; CD11b Antigen; CD18 Antigens; Cell Membrane; Guanidines; Humans; Hydrogen-Ion Concentration; L-Selectin; Lactic Acid; Macrophage-1 Antigen; Metalloproteases; Neutrophils; p38 Mitogen-Activated Protein Kinases; Protease Inhibitors; Protein Kinase Inhibitors; Reperfusion Injury; Sodium-Hydrogen Exchangers; Sulfones; Time Factors

2008