aminooxyacetic acid has been researched along with Myocardial Ischemia in 2 studies
Aminooxyacetic Acid: A compound that inhibits aminobutyrate aminotransferase activity in vivo, thereby raising the level of gamma-aminobutyric acid in tissues.
(aminooxy)acetic acid : A member of the class of hydroxylamines that is acetic acid substituted at postion 2 by an aminooxy group. It is a compound which inhibits aminobutyrate aminotransferase activity in vivo, resulting in increased levels of gamma-aminobutyric acid in tissues.
Myocardial Ischemia: A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION).
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Jespersen, NR | 1 |
Yokota, T | 1 |
Støttrup, NB | 2 |
Bergdahl, A | 1 |
Paelestik, KB | 1 |
Povlsen, JA | 2 |
Dela, F | 1 |
Bøtker, HE | 2 |
Løfgren, B | 1 |
Rasmussen, LE | 1 |
Solskov, L | 1 |
Krarup, PM | 1 |
Kristiansen, SB | 1 |
Nielsen, TT | 1 |
Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
---|---|---|---|---|---|---|---|
Cardiac Mitochondrial Function After Heart Transplantation[NCT04105803] | 64 participants (Anticipated) | Observational | 2019-04-25 | Enrolling by invitation | |||
Cardiac Mitochondrial Function in Explanted Human Hearts[NCT04500938] | 24 participants (Anticipated) | Observational | 2020-08-30 | Not yet recruiting | |||
[information is prepared from clinicaltrials.gov, extracted Sep-2024] |
2 other studies available for aminooxyacetic acid and Myocardial Ischemia
Article | Year |
---|---|
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle | 2017 |
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle | 2017 |
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle | 2017 |
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle | 2017 |
Amino acid transamination is crucial for ischaemic cardioprotection in normal and preconditioned isolated rat hearts--focus on L-glutamate.
Topics: Aminooxyacetic Acid; Animals; Cardiotonic Agents; Glutamic Acid; Heart; In Vitro Techniques; Ischemi | 2010 |