aminooxyacetic acid has been researched along with Kidney Diseases in 8 studies
Aminooxyacetic Acid: A compound that inhibits aminobutyrate aminotransferase activity in vivo, thereby raising the level of gamma-aminobutyric acid in tissues.
(aminooxy)acetic acid : A member of the class of hydroxylamines that is acetic acid substituted at postion 2 by an aminooxy group. It is a compound which inhibits aminobutyrate aminotransferase activity in vivo, resulting in increased levels of gamma-aminobutyric acid in tissues.
Kidney Diseases: Pathological processes of the KIDNEY or its component tissues.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Carbon dioxide absorbents degrade both halothane and sevoflurane to toxic unsaturated compounds (CF2=CBrCl and CH2F-O-C[=CF2][CF3] [i." | 1.30 | Quantitative differences in the production and toxicity of CF2=BrCl versus CH2F-O-C(=CF2)(CF3) (compound A): the safety of halothane does not indicate the safety of sevoflurane. ( Eger, EI; Gong, D; Ionescu, P; Kerschmann, RL; Laster, MJ; Weiskopf, RB, 1997) |
| " In contrast apical treatment with PCBD-NAC was only toxic at high concentrations (greater than 850 microM), and this effect could hardly be inhibited by AOAA." | 1.27 | Differential toxicity as a result of apical and basolateral treatment of LLC-PK1 monolayers with S-(1,2,3,4,4-pentachlorobutadienyl)glutathione and N-acetyl-S-(1,2,3,4,4-pentachlorobutadienyl)-L-cysteine. ( Mertens, JJ; Spenkelink, B; Temmink, JH; van Bladeren, PJ; van Doorn, WJ; Weijnen, JG, 1988) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (25.00) | 18.7374 |
| 1990's | 5 (62.50) | 18.2507 |
| 2000's | 1 (12.50) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Martin, JL | 1 |
| Laster, MJ | 2 |
| Kandel, L | 1 |
| Kerschmann, RL | 2 |
| Reed, GF | 1 |
| Eger, EI | 2 |
| Ionescu, P | 1 |
| Gong, D | 1 |
| Weiskopf, RB | 1 |
| Lock, EA | 1 |
| Ishmael, J | 1 |
| Townsend, DM | 1 |
| Hanigan, MH | 1 |
| Rankin, GO | 1 |
| Shih, HC | 1 |
| Teets, VJ | 1 |
| Yang, DJ | 1 |
| Nicoll, DW | 1 |
| Brown, PI | 1 |
| Monks, TJ | 1 |
| Jones, TW | 1 |
| Hill, BA | 1 |
| Lau, SS | 1 |
| Mertens, JJ | 1 |
| Weijnen, JG | 1 |
| van Doorn, WJ | 1 |
| Spenkelink, B | 1 |
| Temmink, JH | 1 |
| van Bladeren, PJ | 1 |
| Commandeur, JN | 1 |
| Oostendorp, RA | 1 |
| Schoofs, PR | 1 |
| Xu, B | 1 |
| Vermeulen, NP | 1 |
8 other studies available for aminooxyacetic acid and Kidney Diseases
| Article | Year |
|---|---|
Metabolism of compound A by renal cysteine-S-conjugate beta-lyase is not the mechanism of compound A-induced renal injury in the rat.
Topics: Aminooxyacetic Acid; Anesthetics, Inhalation; Animals; Buthionine Sulfoximine; Carbon-Sulfur Lyases; | 1996 |
Quantitative differences in the production and toxicity of CF2=BrCl versus CH2F-O-C(=CF2)(CF3) (compound A): the safety of halothane does not indicate the safety of sevoflurane.
Topics: Absorption; Aminooxyacetic Acid; Anesthetics, Inhalation; Animals; Chemical Phenomena; Chemistry, Ph | 1997 |
The nephrotoxicity and hepatotoxicity of 1,1,2,2-tetrafluoroethyl-L-cysteine in the rat.
Topics: Acetylcysteine; Aminooxyacetic Acid; Animals; Butadienes; Cysteine; Dose-Response Relationship, Drug | 1998 |
Inhibition of gamma-glutamyl transpeptidase or cysteine S-conjugate beta-lyase activity blocks the nephrotoxicity of cisplatin in mice.
Topics: Aminooxyacetic Acid; Animals; Antimetabolites, Antineoplastic; Antineoplastic Agents; Blood Urea Nit | 2002 |
N-(3,5-dichlorophenyl)succinimide nephrotoxicity: evidence against the formation of nephrotoxic glutathione or cysteine conjugates.
Topics: Aminooxyacetic Acid; Animals; Biotransformation; Cysteine; Fungicides, Industrial; gamma-Glutamyltra | 1991 |
Nephrotoxicity of 2-bromo-(cystein-S-yl) hydroquinone and 2-bromo-(N-acetyl-L-cystein-S-yl) hydroquinone thioethers.
Topics: Aminooxyacetic Acid; Animals; Cysteine; Hydroquinones; Kidney; Kidney Diseases; Male; Necrosis; Prob | 1991 |
Differential toxicity as a result of apical and basolateral treatment of LLC-PK1 monolayers with S-(1,2,3,4,4-pentachlorobutadienyl)glutathione and N-acetyl-S-(1,2,3,4,4-pentachlorobutadienyl)-L-cysteine.
Topics: Acetylcysteine; Aminooxyacetic Acid; Animals; Anion Transport Proteins; Anions; Biological Transport | 1988 |
Nephrotoxicity and hepatotoxicity of 1,1-dichloro-2,2-difluoroethylene in the rat. Indications for differential mechanisms of bioactivation.
Topics: Acetates; Acetic Acid; Aminooxyacetic Acid; Animals; Biotransformation; Chemical and Drug Induced Li | 1987 |