aminooxyacetic acid and Heart Disease, Ischemic
aminooxyacetic acid has been researched along with Heart Disease, Ischemic in 2 studies
Aminooxyacetic Acid: A compound that inhibits aminobutyrate aminotransferase activity in vivo, thereby raising the level of gamma-aminobutyric acid in tissues.
(aminooxy)acetic acid : A member of the class of hydroxylamines that is acetic acid substituted at postion 2 by an aminooxy group. It is a compound which inhibits aminobutyrate aminotransferase activity in vivo, resulting in increased levels of gamma-aminobutyric acid in tissues.
Research
Studies (2)
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors
Authors | Studies |
---|---|
Jespersen, NR | 1 |
Yokota, T | 1 |
Støttrup, NB | 2 |
Bergdahl, A | 1 |
Paelestik, KB | 1 |
Povlsen, JA | 2 |
Dela, F | 1 |
Bøtker, HE | 2 |
Løfgren, B | 1 |
Rasmussen, LE | 1 |
Solskov, L | 1 |
Krarup, PM | 1 |
Kristiansen, SB | 1 |
Nielsen, TT | 1 |
Clinical Trials (2)
Trial Overview
Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
---|---|---|---|---|---|---|---|
Cardiac Mitochondrial Function After Heart Transplantation[NCT04105803] | 64 participants (Anticipated) | Observational | 2019-04-25 | Enrolling by invitation | |||
Cardiac Mitochondrial Function in Explanted Human Hearts[NCT04500938] | 24 participants (Anticipated) | Observational | 2020-08-30 | Not yet recruiting | |||
[information is prepared from clinicaltrials.gov, extracted Sep-2024] |
Other Studies
2 other studies available for aminooxyacetic acid and Heart Disease, Ischemic
Article | Year |
---|---|
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle | 2017 |
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle | 2017 |
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle | 2017 |
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle | 2017 |
Amino acid transamination is crucial for ischaemic cardioprotection in normal and preconditioned isolated rat hearts--focus on L-glutamate.
Topics: Aminooxyacetic Acid; Animals; Cardiotonic Agents; Glutamic Acid; Heart; In Vitro Techniques; Ischemi | 2010 |