Page last updated: 2024-10-16

aminooxyacetic acid and Heart Disease, Ischemic

aminooxyacetic acid has been researched along with Heart Disease, Ischemic in 2 studies

Aminooxyacetic Acid: A compound that inhibits aminobutyrate aminotransferase activity in vivo, thereby raising the level of gamma-aminobutyric acid in tissues.
(aminooxy)acetic acid : A member of the class of hydroxylamines that is acetic acid substituted at postion 2 by an aminooxy group. It is a compound which inhibits aminobutyrate aminotransferase activity in vivo, resulting in increased levels of gamma-aminobutyric acid in tissues.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Jespersen, NR1
Yokota, T1
Støttrup, NB2
Bergdahl, A1
Paelestik, KB1
Povlsen, JA2
Dela, F1
Bøtker, HE2
Løfgren, B1
Rasmussen, LE1
Solskov, L1
Krarup, PM1
Kristiansen, SB1
Nielsen, TT1

Clinical Trials (2)

Trial Overview

TrialPhaseEnrollmentStudy TypeStart DateStatus
Cardiac Mitochondrial Function After Heart Transplantation[NCT04105803]64 participants (Anticipated)Observational2019-04-25Enrolling by invitation
Cardiac Mitochondrial Function in Explanted Human Hearts[NCT04500938]24 participants (Anticipated)Observational2020-08-30Not yet recruiting
[information is prepared from clinicaltrials.gov, extracted Sep-2024]

Other Studies

2 other studies available for aminooxyacetic acid and Heart Disease, Ischemic

ArticleYear
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
    The Journal of physiology, 2017, 06-15, Volume: 595, Issue:12

    Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle

2017
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
    The Journal of physiology, 2017, 06-15, Volume: 595, Issue:12

    Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle

2017
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
    The Journal of physiology, 2017, 06-15, Volume: 595, Issue:12

    Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle

2017
Pre-ischaemic mitochondrial substrate constraint by inhibition of malate-aspartate shuttle preserves mitochondrial function after ischaemia-reperfusion.
    The Journal of physiology, 2017, 06-15, Volume: 595, Issue:12

    Topics: Aminooxyacetic Acid; Animals; Aspartic Acid; Cardiotonic Agents; Cell Respiration; Citric Acid Cycle

2017
Amino acid transamination is crucial for ischaemic cardioprotection in normal and preconditioned isolated rat hearts--focus on L-glutamate.
    Experimental physiology, 2010, Volume: 95, Issue:1

    Topics: Aminooxyacetic Acid; Animals; Cardiotonic Agents; Glutamic Acid; Heart; In Vitro Techniques; Ischemi

2010
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