Page last updated: 2024-08-21

aminoimidazole carboxamide and Vascular Diseases

aminoimidazole carboxamide has been researched along with Vascular Diseases in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	1 (50.00)	29.6817
2010's	1 (50.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Chen, K; Daiber, A; Dopheide, J; Keaney, JF; Münzel, T; Schuhmacher, S; Schulz, E; Thomas, SR; Wenzel, P	1
Chen, K; Croft, KD; Keaney, JF; Li, C; Ward, NC	1

Other Studies

2 other study(ies) available for aminoimidazole carboxamide and Vascular Diseases

Article	Year
Suppression of the JNK pathway by induction of a metabolic stress response prevents vascular injury and dysfunction. Circulation, 2008, Sep-23, Volume: 118, Issue:13 Topics: Adaptation, Physiological; Adenylate Kinase; Aminoimidazole Carboxamide; Animals; Cell Death; Chlorocebus aethiops; COS Cells; Endothelial Cells; Heat-Shock Proteins; Humans; Hydrogen Peroxide; JNK Mitogen-Activated Protein Kinases; Male; Membrane Potential, Mitochondrial; Mice; Mice, Inbred C57BL; Mutagenesis; Oxidants; Oxidative Stress; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha; Ribonucleotides; RNA, Small Interfering; Transcription Factors; Umbilical Veins; Vascular Diseases	2008
Chronic activation of AMP-activated protein kinase prevents 20-hydroxyeicosatetraenoic acid-induced endothelial dysfunction. Clinical and experimental pharmacology & physiology, 2011, Volume: 38, Issue:5 Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Aorta; Cells, Cultured; Cytoprotection; Drug Evaluation, Preclinical; Endothelium, Vascular; Enzyme Activators; Humans; Hydroxyeicosatetraenoic Acids; Male; Mice; Mice, Inbred C57BL; Nitric Oxide Synthase Type III; Phosphorylation; Proto-Oncogene Proteins c-akt; Ribonucleotides; Time Factors; Vascular Diseases; Vasoconstrictor Agents	2011

Article

Year

Suppression of the JNK pathway by induction of a metabolic stress response prevents vascular injury and dysfunction.

Circulation, 2008, Sep-23, Volume: 118, Issue:13

Topics: Adaptation, Physiological; Adenylate Kinase; Aminoimidazole Carboxamide; Animals; Cell Death; Chlorocebus aethiops; COS Cells; Endothelial Cells; Heat-Shock Proteins; Humans; Hydrogen Peroxide; JNK Mitogen-Activated Protein Kinases; Male; Membrane Potential, Mitochondrial; Mice; Mice, Inbred C57BL; Mutagenesis; Oxidants; Oxidative Stress; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha; Ribonucleotides; RNA, Small Interfering; Transcription Factors; Umbilical Veins; Vascular Diseases

2008

Chronic activation of AMP-activated protein kinase prevents 20-hydroxyeicosatetraenoic acid-induced endothelial dysfunction.

Clinical and experimental pharmacology & physiology, 2011, Volume: 38, Issue:5

Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Aorta; Cells, Cultured; Cytoprotection; Drug Evaluation, Preclinical; Endothelium, Vascular; Enzyme Activators; Humans; Hydroxyeicosatetraenoic Acids; Male; Mice; Mice, Inbred C57BL; Nitric Oxide Synthase Type III; Phosphorylation; Proto-Oncogene Proteins c-akt; Ribonucleotides; Time Factors; Vascular Diseases; Vasoconstrictor Agents

2011