aminoimidazole carboxamide has been researched along with Muscular Dystrophy, Duchenne in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Al-Rewashdy, H; Jasmin, BJ; Lin, W; Ljubicic, V; Renaud, JM | 1 |
| Ahmed, A; Amirouche, A; Bronicki, LM; Crawford Parks, TE; Jasmin, BJ; Ljubicic, V; Péladeau, C; Renaud, JM | 1 |
| Jasmin, BJ; Khogali, S; Ljubicic, V; Renaud, JM | 1 |
3 other study(ies) available for aminoimidazole carboxamide and Muscular Dystrophy, Duchenne
| Article | Year |
|---|---|
Utrophin A is essential in mediating the functional adaptations of mdx mouse muscle following chronic AMPK activation.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Disease Models, Animal; Dystroglycans; Female; Genotyping Techniques; Hand Strength; Male; Mice; Mice, Inbred mdx; Mice, Knockout; Muscular Dystrophy, Duchenne; Myofibrils; Phenotype; Ribonucleosides; Utrophin | 2015 |
Combinatorial therapeutic activation with heparin and AICAR stimulates additive effects on utrophin A expression in dystrophic muscles.
Topics: Aminoimidazole Carboxamide; Animals; Cell Line; Drug Therapy, Combination; Heparin; Mice; Mice, Inbred mdx; Muscle, Skeletal; Muscular Dystrophy, Duchenne; p38 Mitogen-Activated Protein Kinases; Ribonucleotides; Signal Transduction; Up-Regulation; Utrophin | 2016 |
Chronic AMPK stimulation attenuates adaptive signaling in dystrophic skeletal muscle.
Topics: Adaptation, Physiological; Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Disease Models, Animal; Mice; Mice, Inbred C57BL; Mice, Inbred mdx; Mice, Transgenic; Muscle, Skeletal; Muscular Dystrophy, Duchenne; Physical Conditioning, Animal; Ribonucleotides; Signal Transduction | 2012 |