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aminoimidazole carboxamide and Leukemia, Myelogenous, Chronic, BCR-ABL Positive

aminoimidazole carboxamide has been researched along with Leukemia, Myelogenous, Chronic, BCR-ABL Positive in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's1 (33.33)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Liang, HT; Liu, W; Tang, L; You, Y; Zhu, XJ; Zhu, XY; Zou, P1
Auberger, P; Belhacene, N; Ben Sahra, I; Bost, F; Cassuto, JP; Colosetti, P; Gounon, P; Hofman, P; Puissant, A; Robert, G1
Altman, JK; Donato, NJ; Glaser, H; Platanias, LC; Vakana, E1

Other Studies

3 other study(ies) available for aminoimidazole carboxamide and Leukemia, Myelogenous, Chronic, BCR-ABL Positive

ArticleYear
AICAR and Decitabine Enhance the Sensitivity of K562 Cells to Imatinib by Promoting Mitochondrial Activity.
    Current medical science, 2020, Volume: 40, Issue:5

    Topics: Aminoimidazole Carboxamide; Apoptosis; Cell Line, Tumor; Cell Proliferation; Decitabine; Drug Resistance, Neoplasm; Fusion Proteins, bcr-abl; Humans; Imatinib Mesylate; K562 Cells; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Mitochondria; Protein Kinase Inhibitors; Ribonucleotides

2020
Acadesine kills chronic myelogenous leukemia (CML) cells through PKC-dependent induction of autophagic cell death.
    PloS one, 2009, Nov-18, Volume: 4, Issue:11

    Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Apoptosis; Autophagy; Cell Line, Tumor; Cell Proliferation; Humans; K562 Cells; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Mice; Mice, Nude; Protein Kinase C; Ribonucleosides; RNA, Small Interfering; Translocation, Genetic

2009
Antileukemic effects of AMPK activators on BCR-ABL-expressing cells.
    Blood, 2011, Dec-08, Volume: 118, Issue:24

    Topics: Amino Acid Substitution; Aminoimidazole Carboxamide; AMP-Activated Protein Kinase Kinases; Antineoplastic Agents; Apoptosis; Bone Marrow Cells; Cell Line, Transformed; Cell Survival; Cell Transformation, Neoplastic; Down-Regulation; Drug Resistance, Neoplasm; Enzyme Activation; Fusion Proteins, bcr-abl; Hematopoietic Stem Cells; Humans; Leukemia, Lymphoid; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Metformin; Molecular Targeted Therapy; Mutant Proteins; Prodrugs; Protein Kinases; Recombinant Proteins; Ribonucleotides

2011