aminoimidazole carboxamide has been researched along with Glial Cell Tumors in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 1 (25.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chang, C; Chornenkyy, Y; Hawkins, C; Joshi, S; Liu, J; Ryall, S; Shen, H; Siddaway, R; Tsoli, M; Yu, M; Ziegler, DS | 1 |
| Chang, CY; Chen, CJ; Chen, WY; Huang, HY; Kuan, YH; Li, JR; Ou, YC; Pan, PH; Wu, CC | 1 |
| Harhaji-Trajkovic, L; Isenovic, E; Janjetovic, K; Micic, D; Misirkic, M; Prica, M; Stevanovic, D; Sudar, E; Sumarac-Dumanovic, M; Trajkovic, V; Vucicevic, L | 1 |
| Bauer, DE; Buzzai, M; Deberardinis, RJ; Elstrom, RL; Hatzivassiliou, G; Jones, RG; Thompson, CB | 1 |
4 other study(ies) available for aminoimidazole carboxamide and Glial Cell Tumors
| Article | Year |
|---|---|
Targeting reduced mitochondrial DNA quantity as a therapeutic approach in pediatric high-grade gliomas.
Topics: Aminoimidazole Carboxamide; Animals; Brain Neoplasms; Child; Dichloroacetic Acid; DNA, Mitochondrial; Energy Metabolism; Gene Dosage; Glioma; Glycolysis; Humans; Mice; Ribonucleotides; Xenograft Model Antitumor Assays | 2020 |
Autophagy contributes to gefitinib-induced glioma cell growth inhibition.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Apoptosis Regulatory Proteins; Autophagy; Cell Line, Tumor; Gefitinib; Glioma; Growth Inhibitors; Humans; Mice; Mice, Inbred BALB C; Mice, Nude; Microtubule-Associated Proteins; Proto-Oncogene Proteins c-akt; Quinazolines; Ribonucleotides; Signal Transduction; TOR Serine-Threonine Kinases | 2014 |
AMP-activated protein kinase-dependent and -independent mechanisms underlying in vitro antiglioma action of compound C.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Antineoplastic Agents; Antioxidants; Apoptosis; bcl-2-Associated X Protein; Caspases; Cell Cycle; Cell Death; Cell Division; Cell Line, Tumor; Enzyme Activation; G2 Phase; Glioma; Humans; Metformin; Pheochromocytoma; Proto-Oncogene Proteins c-bcl-2; Pyrazoles; Pyrimidines; Reactive Oxygen Species; Reverse Transcriptase Polymerase Chain Reaction; Ribonucleotides | 2009 |
The glucose dependence of Akt-transformed cells can be reversed by pharmacologic activation of fatty acid beta-oxidation.
Topics: Aminoimidazole Carboxamide; Cell Survival; Fatty Acids; Glioma; Glucose; Humans; Hypoglycemic Agents; Oxidation-Reduction; Protein Serine-Threonine Kinases; Protein-Tyrosine Kinases; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-akt; Ribonucleotides; Tumor Cells, Cultured | 2005 |