aminoimidazole carboxamide has been researched along with Age-Related Memory Disorders in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 4 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Cao, Y; Li, M; Li, MZ; Liu, BJ; Shi, FX; Sun, DS; Tian, Q; Wang, JZ; Wang, L; Xu, WQ; Zhou, XW | 1 |
Chai, DM; Du, LL; Li, XH; Liu, LB; Liu, R; Wang, JZ; Wu, K; Zhang, FC; Zhang, HB; Zhao, LN; Zhou, XW | 1 |
Kim, DM; Leem, YH | 1 |
Dash, PK; Hill, JL; Hylin, MJ; Kobori, N; Moore, AN; Rozas, NS; Zhao, J | 1 |
4 other study(ies) available for aminoimidazole carboxamide and Age-Related Memory Disorders
Article | Year |
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Deletion of Type-2 Cannabinoid Receptor Induces Alzheimer's Disease-Like Tau Pathology and Memory Impairment Through AMPK/GSK3β Pathway.
Topics: Adenylate Kinase; Aging; Alzheimer Disease; Aminoimidazole Carboxamide; Animals; Cannabinoids; Enzyme Activation; Gene Deletion; Glycogen Synthase Kinase 3 beta; Hippocampus; Memory; Memory Disorders; Mice, Inbred C57BL; Mice, Transgenic; Mitochondria; Phosphorylation; Receptor, Cannabinoid, CB2; Resveratrol; Ribonucleotides; Signal Transduction; tau Proteins | 2018 |
AMPK activation ameliorates Alzheimer's disease-like pathology and spatial memory impairment in a streptozotocin-induced Alzheimer's disease model in rats.
Topics: Adenylate Kinase; Alzheimer Disease; Aminoimidazole Carboxamide; Animals; Caspase 3; Disease Models, Animal; Hippocampus; Hypoglycemic Agents; Male; Memory Disorders; Phosphorylation; Rats; Rats, Sprague-Dawley; Ribonucleotides; Spatial Memory; Streptozocin; tau Proteins | 2015 |
Chronic stress-induced memory deficits are reversed by regular exercise via AMPK-mediated BDNF induction.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Brain-Derived Neurotrophic Factor; Cells, Cultured; Chronic Disease; Disease Models, Animal; Dose-Response Relationship, Drug; Exercise Therapy; Hippocampus; Male; Maze Learning; Memory Disorders; Mice, Inbred C57BL; Neurogenesis; Nootropic Agents; Restraint, Physical; Ribonucleotides; Running; Stress, Psychological | 2016 |
Traumatic brain injury decreases AMP-activated protein kinase activity and pharmacological enhancement of its activity improves cognitive outcome.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Brain Injuries, Traumatic; Enzyme Activators; Hippocampus; Male; Maze Learning; Memory Disorders; Metformin; Mice; Mice, Inbred C57BL; Neuroprotective Agents; Nootropic Agents; Parietal Lobe; Phosphorylation; Psychomotor Performance; Rats; Rats, Sprague-Dawley; Ribonucleotides | 2016 |