alphaxalone has been researched along with Epilepsy, Absence in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 3 (100.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Banerjee, PK; Brailowsky, S; Olsen, RW; Snead, OC; Tillakaratne, NJ; Tobin, AJ | 1 |
| Banerjee, PK; Snead, OC | 1 |
| Banerjee, PK; Liu, CC; Snead, OC | 1 |
3 other study(ies) available for alphaxalone and Epilepsy, Absence
| Article | Year |
|---|---|
Absence seizures decrease steroid modulation of t-[35S]butylbicyclophosphorothionate binding in thalamic relay nuclei.
Topics: Action Potentials; Animals; Binding Sites; Bridged Bicyclo Compounds, Heterocyclic; Desoxycorticosterone; Epilepsy, Absence; gamma-Aminobutyric Acid; Hydroxybutyrates; Male; Pentobarbital; Pregnanediones; Rats; Rats, Sprague-Dawley; Sulfur Radioisotopes; Thalamic Nuclei | 1998 |
Neuroactive steroids exacerbate gamma-hydroxybutyric acid-induced absence seizures in rats.
Topics: Anesthetics; Animals; Desoxycorticosterone; Epilepsy, Absence; Hydroxybutyrates; Male; Pregnanediones; Rats; Rats, Sprague-Dawley; Thalamic Nuclei | 1998 |
Steroid-inhibition of [3H]gamma-hydroxybutyric acid (GHB) binding in thalamic relay nuclei increases during absence seizures.
Topics: Anesthetics; Animals; Binding, Competitive; Cerebral Cortex; Desoxycorticosterone; Epilepsy, Absence; gamma-Aminobutyric Acid; Male; Neurons; Parietal Lobe; Pregnanediones; Rats; Rats, Sprague-Dawley; Receptors, GABA-A; Thalamus; Tritium; Up-Regulation | 1998 |