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aloe emodin and Cell Transformation, Neoplastic

aloe emodin has been researched along with Cell Transformation, Neoplastic in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Dong, Z; Guo, L; Liu, K; Zhang, J; Zhang, Q1
Beppu, H; Chihara, T; Kaneko, T; Shimpo, K; Shinzato, M; Sonoda, S; Wakamatsu, K; Yukitake, J1
Acevedo-Duncan, M; Patel, R; Patel, S; Russell, C1

Other Studies

3 other study(ies) available for aloe emodin and Cell Transformation, Neoplastic

ArticleYear
Aloe emodin suppresses EGF‑induced neoplastic cell transformation by inhibiting the ERK/MSK1 and AKT/GSK3β signaling pathways.
    Molecular medicine reports, 2018, Volume: 18, Issue:6

    Topics: Anthraquinones; Antineoplastic Agents; Cell Transformation, Neoplastic; Cyclin D1; Dose-Response Relationship, Drug; Epidermal Growth Factor; Extracellular Signal-Regulated MAP Kinases; Gene Expression Regulation; Glycogen Synthase Kinase 3 beta; Proto-Oncogene Proteins c-akt; Ribosomal Protein S6 Kinases, 90-kDa; Signal Transduction

2018
Inhibitory effects of low-dose aloe-emodin on the development of colorectal tumors in min mice.
    Asian Pacific journal of cancer prevention : APJCP, 2014, Volume: 15, Issue:14

    Topics: Animals; Anthraquinones; Antineoplastic Agents; Cell Proliferation; Cell Transformation, Neoplastic; Chemoprevention; Colitis; Colorectal Neoplasms; Cyclooxygenase 2; Dextran Sulfate; Dietary Supplements; Female; Intestinal Mucosa; Lipids; Male; Mice; Mice, Inbred C57BL; Nitric Oxide Synthase Type II; RNA, Messenger

2014
Aloe-emodin modulates PKC isozymes, inhibits proliferation, and induces apoptosis in U-373MG glioma cells.
    International immunopharmacology, 2004, Dec-20, Volume: 4, Issue:14

    Topics: Anthraquinones; Apoptosis; Blotting, Western; Brain Neoplasms; Cell Cycle; Cell Fractionation; Cell Line, Tumor; Cell Proliferation; Cell Survival; Cell Transformation, Neoplastic; DNA; Emodin; Glioma; Humans; Isoenzymes; Neuroglia; Protein Kinase C; S Phase

2004