allysine has been researched along with Amino Acid Metabolism Disorders, Inborn in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (50.00) | 24.3611 |
| 2020's | 2 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, H; DeVita, RJ; Dodatko, T; Houten, SM; Leandro, J; Stauffer, B; Yu, C | 1 |
| Bouchereau, J; Schiff, M | 1 |
| Hoffmann, GF; Koeller, DM; Kölker, S; Okun, JG; Opp, S; Sauer, SW | 1 |
| Ceyhan, S; Clayton, PT; Footitt, EJ; Jakobs, C; Mills, PB; Struys, EA; Waters, PJ | 1 |
1 review(s) available for allysine and Amino Acid Metabolism Disorders, Inborn
| Article | Year |
|---|---|
Inherited Disorders of Lysine Metabolism: A Review.
Topics: 2-Aminoadipic Acid; Aldehyde Dehydrogenase; Amino Acid Metabolism, Inborn Errors; Arginine; Brain; Brain Diseases, Metabolic; Brain Diseases, Metabolic, Inborn; Carnitine; Epilepsy; Glutarates; Glutaryl-CoA Dehydrogenase; Humans; Lysine; Metabolic Diseases; Pyridoxal Phosphate; Pyridoxine | 2020 |
3 other study(ies) available for allysine and Amino Acid Metabolism Disorders, Inborn
| Article | Year |
|---|---|
Deletion of 2-aminoadipic semialdehyde synthase limits metabolite accumulation in cell and mouse models for glutaric aciduria type 1.
Topics: 2-Aminoadipic Acid; Amino Acid Metabolism, Inborn Errors; Animals; Brain; Brain Diseases, Metabolic; CRISPR-Cas Systems; Disease Models, Animal; Female; Glutarates; Glutaryl-CoA Dehydrogenase; HEK293 Cells; Humans; Liver; Male; Mice; Mice, Knockout | 2020 |
Therapeutic modulation of cerebral L-lysine metabolism in a mouse model for glutaric aciduria type I.
Topics: 2-Aminoadipate Transaminase; 2-Aminoadipic Acid; Amino Acid Metabolism, Inborn Errors; Analysis of Variance; Animals; Arginine; Brain; Brain Diseases, Metabolic; Carnitine; Catalase; Glutaryl-CoA Dehydrogenase; Ketoglutaric Acids; Lysine; Mice | 2011 |
Urinary AASA excretion is elevated in patients with molybdenum cofactor deficiency and isolated sulphite oxidase deficiency.
Topics: 2-Aminoadipic Acid; Adolescent; Amino Acid Metabolism, Inborn Errors; Child; Coenzymes; Cysteine; Humans; Infant, Newborn; L-Aminoadipate-Semialdehyde Dehydrogenase; Lysine; Metabolic Networks and Pathways; Metal Metabolism, Inborn Errors; Metalloproteins; Models, Biological; Molybdenum Cofactors; Molybdoferredoxin; Oxidoreductases Acting on Sulfur Group Donors; Pteridines; Sulfite Oxidase; Sulfites | 2012 |