allyl isothiocyanate has been researched along with Prostatic Neoplasms in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Hershberger, P; Johnson, CS; Lew, KL; Singh, SV; Srivastava, SK; Trump, DL; Xiao, D; Zeng, Y | 1 |
| Hershberger, P; Johnson, CS; Kokkinakis, DM; Lew, KL; Singh, SV; Srivastava, SK; Trump, DL; Xiao, D | 1 |
| Gopalkrishnan, A; Keum, YS; Kim, JH; Kong, AN; Nair, S; Shen, G; Xu, C; Yuan, X | 1 |
3 other study(ies) available for allyl isothiocyanate and Prostatic Neoplasms
| Article | Year |
|---|---|
Allyl isothiocyanate, a constituent of cruciferous vegetables, inhibits proliferation of human prostate cancer cells by causing G2/M arrest and inducing apoptosis.
Topics: Apoptosis; bcl-X Protein; Blotting, Western; CDC2 Protein Kinase; cdc25 Phosphatases; Cell Cycle Proteins; Cell Division; Cyclin B; Cyclin B1; Epithelial Cells; G2 Phase; Humans; Isothiocyanates; Male; Mitosis; Prostate; Prostatic Neoplasms; Proto-Oncogene Proteins c-bcl-2; Tumor Cells, Cultured; Vegetables | 2003 |
Allyl isothiocyanate, a constituent of cruciferous vegetables, inhibits growth of PC-3 human prostate cancer xenografts in vivo.
Topics: Animals; Antineoplastic Agents; Apoptosis; Blotting, Western; Cell Cycle Proteins; Dose-Response Relationship, Drug; Genes, bcl-2; Humans; Isothiocyanates; Male; Mice; Mitosis; Neoplasm Transplantation; Prostate; Prostatic Neoplasms; Signal Transduction; Transplantation, Heterologous; Tumor Cells, Cultured; Vegetables | 2003 |
ERK and JNK signaling pathways are involved in the regulation of activator protein 1 and cell death elicited by three isothiocyanates in human prostate cancer PC-3 cells.
Topics: Anticarcinogenic Agents; Apoptosis; Extracellular Signal-Regulated MAP Kinases; Food Preservatives; Humans; Isothiocyanates; JNK Mitogen-Activated Protein Kinases; Male; Prostatic Neoplasms; Signal Transduction; Sulfoxides; Thiocyanates; Transcription Factor AP-1; Transcription, Genetic; Transfection; Tumor Cells, Cultured | 2006 |