allicin has been researched along with Chronic Lung Injury in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Cheng, A; Qiu, M; Shen, N; Zang, G | 1 |
| Ashry, NA; Gameil, NM; Suddek, GM | 1 |
2 other study(ies) available for allicin and Chronic Lung Injury
| Article | Year |
|---|---|
Allicin Improves Lung Injury Induced by Sepsis via Regulation of the Toll-Like Receptor 4 (TLR4)/Myeloid Differentiation Primary Response 88 (MYD88)/Nuclear Factor kappa B (NF-κB) Pathway.
Topics: Acute Lung Injury; Animals; Cell Differentiation; Cytokines; Disulfides; Interleukin-1beta; Lipopolysaccharides; Lung; Lung Injury; Male; Myeloid Differentiation Factor 88; NF-kappa B; Rats; Rats, Sprague-Dawley; Sepsis; Signal Transduction; Sulfinic Acids; Toll-Like Receptor 4; Tumor Necrosis Factor-alpha | 2019 |
Modulation of cyclophosphamide-induced early lung injury by allicin.
Topics: Administration, Oral; Animals; Antineoplastic Agents, Alkylating; Antioxidants; Biomarkers; Cyclophosphamide; Disulfides; Free Radical Scavengers; Injections, Intraperitoneal; Lipid Peroxidation; Lung Injury; Male; Rats; Rats, Sprague-Dawley; Sulfinic Acids; Superoxide Dismutase; Tumor Necrosis Factor-alpha | 2013 |