aldosterone has been researched along with Glucose Intolerance in 12 studies
Glucose Intolerance: A pathological state in which BLOOD GLUCOSE level is less than approximately 140 mg/100 ml of PLASMA at fasting, and above approximately 200 mg/100 ml plasma at 30-, 60-, or 90-minute during a GLUCOSE TOLERANCE TEST. This condition is seen frequently in DIABETES MELLITUS, but also occurs with other diseases and MALNUTRITION.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Impaired glucose tolerance (IGT) is associated with cardiovascular risk factors, but the effects of pioglitazone and metformin on IGT are not well described." | 9.12 | Comparison of the effects of pioglitazone and metformin on insulin resistance and hormonal markers in patients with impaired glucose tolerance and early diabetes. ( Eguchi, K; Fukuda, T; Hoshide, S; Ishikawa, J; Kario, K; Numao, T; Shimada, K; Tomizawa, H, 2007) |
| "After the treatment of primary aldosteronism, glucose intolerance was improved in 66." | 5.56 | Different pathogenesis of glucose intolerance in two subtypes of primary aldosteronism: Aldosterone-producing adenoma and idiopathic hyperaldosteronism. ( Fukuda, I; Moriya, A; Nagao, M; Oikawa, S; Okazaki-Hada, M; Sugihara, H, 2020) |
| "Impaired glucose tolerance (IGT) is associated with cardiovascular risk factors, but the effects of pioglitazone and metformin on IGT are not well described." | 5.12 | Comparison of the effects of pioglitazone and metformin on insulin resistance and hormonal markers in patients with impaired glucose tolerance and early diabetes. ( Eguchi, K; Fukuda, T; Hoshide, S; Ishikawa, J; Kario, K; Numao, T; Shimada, K; Tomizawa, H, 2007) |
| "These results demonstrates that hypertension and insulin resistance induced by COC is associated with increased cardiac RAS and PAI-1 gene expression, which is likely to be through corticosterone-dependent but not aldosterone-dependent mechanism." | 3.85 | Activation of cardiac renin-angiotensin system and plasminogen activator inhibitor-1 gene expressions in oral contraceptive-induced cardiometabolic disorder. ( Kim, IK; Olatunji, LA; Seok, YM; Usman, TO, 2017) |
| "Patients with primary aldosteronism can have impaired pancreatic insulin release and reduction in insulin sensitivity." | 2.42 | The effect of aldosterone on glucose metabolism. ( Corry, DB; Tuck, ML, 2003) |
| "After the treatment of primary aldosteronism, glucose intolerance was improved in 66." | 1.56 | Different pathogenesis of glucose intolerance in two subtypes of primary aldosteronism: Aldosterone-producing adenoma and idiopathic hyperaldosteronism. ( Fukuda, I; Moriya, A; Nagao, M; Oikawa, S; Okazaki-Hada, M; Sugihara, H, 2020) |
| "Aldosterone treatment impaired the rate of glucose uptake, oxidation, and insulin signal transduction in the gastrocnemius muscle through defective expression of IR, IRS-1, Akt, AS160, and GLUT4 genes." | 1.39 | Excess aldosterone-induced changes in insulin signaling molecules and glucose oxidation in gastrocnemius muscle of adult male rat. ( Balasubramanian, K; Mayilvanan, C; Sathish, S; Selvaraj, J, 2013) |
| "Primary hyperaldosteronism (PH), resulting in hypokalaemic hypertension, may be due to an aldosterone-producing adenoma (APA) or bilateral zona glomerulosa hyperplasia." | 1.34 | Prospective evaluation and characteristics of patients with suspected primary hyperaldosteronism. ( Boyne, MS; McKenzie, CA; Wright-Pascoe, R, 2007) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 4 (33.33) | 29.6817 |
| 2010's | 6 (50.00) | 24.3611 |
| 2020's | 2 (16.67) | 2.80 |
| Authors | Studies |
|---|---|
| Adegoke, TE | 1 |
| Sabinari, IW | 1 |
| Usman, TO | 2 |
| Abdulkareem, TO | 1 |
| Michael, OS | 1 |
| Adeyanju, OA | 1 |
| Dibia, C | 1 |
| Omotoye, OO | 1 |
| Oyabambi, AO | 1 |
| Olatunji, LA | 2 |
| Okazaki-Hada, M | 1 |
| Moriya, A | 1 |
| Nagao, M | 1 |
| Oikawa, S | 1 |
| Fukuda, I | 1 |
| Sugihara, H | 1 |
| Sherajee, SJ | 1 |
| Rafiq, K | 1 |
| Nakano, D | 1 |
| Mori, H | 1 |
| Kobara, H | 1 |
| Hitomi, H | 1 |
| Fujisawa, Y | 1 |
| Kobori, H | 1 |
| Masaki, T | 1 |
| Nishiyama, A | 1 |
| Armani, A | 1 |
| Cinti, F | 1 |
| Marzolla, V | 1 |
| Morgan, J | 1 |
| Cranston, GA | 1 |
| Antelmi, A | 1 |
| Carpinelli, G | 1 |
| Canese, R | 1 |
| Pagotto, U | 1 |
| Quarta, C | 1 |
| Malorni, W | 1 |
| Matarrese, P | 1 |
| Marconi, M | 1 |
| Fabbri, A | 1 |
| Rosano, G | 1 |
| Cinti, S | 1 |
| Young, MJ | 1 |
| Caprio, M | 1 |
| Seok, YM | 1 |
| Kim, IK | 1 |
| Mendez, N | 1 |
| Halabi, D | 1 |
| Spichiger, C | 1 |
| Salazar, ER | 1 |
| Vergara, K | 1 |
| Alonso-Vasquez, P | 1 |
| Carmona, P | 1 |
| Sarmiento, JM | 1 |
| Richter, HG | 1 |
| Seron-Ferre, M | 1 |
| Torres-Farfan, C | 1 |
| Michailidou, Z | 1 |
| Carter, RN | 1 |
| Marshall, E | 1 |
| Sutherland, HG | 1 |
| Brownstein, DG | 1 |
| Owen, E | 1 |
| Cockett, K | 1 |
| Kelly, V | 1 |
| Ramage, L | 1 |
| Al-Dujaili, EA | 1 |
| Ross, M | 1 |
| Maraki, I | 1 |
| Newton, K | 1 |
| Holmes, MC | 1 |
| Seckl, JR | 1 |
| Morton, NM | 1 |
| Kenyon, CJ | 1 |
| Chapman, KE | 1 |
| Selvaraj, J | 1 |
| Sathish, S | 1 |
| Mayilvanan, C | 1 |
| Balasubramanian, K | 1 |
| Saha, S | 1 |
| Schwarz, PE | 1 |
| Bergmann, S | 1 |
| Bornstein, SR | 1 |
| Graessler, J | 1 |
| Kopprasch, S | 1 |
| Corry, DB | 1 |
| Tuck, ML | 1 |
| Eguchi, K | 1 |
| Tomizawa, H | 1 |
| Ishikawa, J | 1 |
| Hoshide, S | 1 |
| Numao, T | 1 |
| Fukuda, T | 1 |
| Shimada, K | 1 |
| Kario, K | 1 |
| McKenzie, CA | 1 |
| Wright-Pascoe, R | 1 |
| Boyne, MS | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Metabolic Syndrome and Insulin Resistance in Primary Aldosteronism[NCT00173082] | 100 participants (Anticipated) | Observational | 2005-06-30 | Recruiting | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
1 review available for aldosterone and Glucose Intolerance
| Article | Year |
|---|---|
The effect of aldosterone on glucose metabolism.
Topics: Aldosterone; Diabetes Mellitus; Glucose; Glucose Intolerance; Humans; Hyperaldosteronism; Insulin Re | 2003 |
2 trials available for aldosterone and Glucose Intolerance
| Article | Year |
|---|---|
Circulating very-low-density lipoprotein from subjects with impaired glucose tolerance accelerates adrenocortical cortisol and aldosterone synthesis.
Topics: Adrenal Cortex; Aged; Aldosterone; Cell Line; Glucose Intolerance; Glycated Hemoglobin; Humans; Hydr | 2013 |
Comparison of the effects of pioglitazone and metformin on insulin resistance and hormonal markers in patients with impaired glucose tolerance and early diabetes.
Topics: Adiponectin; Aged; Aldosterone; Biomarkers; Blood Glucose; Cross-Over Studies; Diabetes Mellitus; Fa | 2007 |
9 other studies available for aldosterone and Glucose Intolerance
| Article | Year |
|---|---|
Allopurinol and valproic acid improve cardiac triglyceride and Na
Topics: Adenosine Triphosphatases; Aldosterone; Allopurinol; Animals; Contraceptives, Oral, Combined; Female | 2022 |
Different pathogenesis of glucose intolerance in two subtypes of primary aldosteronism: Aldosterone-producing adenoma and idiopathic hyperaldosteronism.
Topics: Adenoma; Aldosterone; Biomarkers; Female; Follow-Up Studies; Glucose Intolerance; Humans; Hyperaldos | 2020 |
Aldosterone aggravates glucose intolerance induced by high fructose.
Topics: Aldosterone; Animals; Blood Glucose; Fructose; Glucose Intolerance; Glucose Tolerance Test; Immediat | 2013 |
Mineralocorticoid receptor antagonism induces browning of white adipose tissue through impairment of autophagy and prevents adipocyte dysfunction in high-fat-diet-fed mice.
Topics: Adipocytes; Adipogenesis; Adipose Tissue, Brown; Adipose Tissue, White; Aldosterone; Androstenes; An | 2014 |
Activation of cardiac renin-angiotensin system and plasminogen activator inhibitor-1 gene expressions in oral contraceptive-induced cardiometabolic disorder.
Topics: Aldosterone; Animals; Ataxia Telangiectasia Mutated Proteins; Cardiovascular Diseases; Contraceptive | 2017 |
Gestational Chronodisruption Impairs Circadian Physiology in Rat Male Offspring, Increasing the Risk of Chronic Disease.
Topics: Aldosterone; Animals; Blood Pressure; Body Temperature; Chronic Disease; Circadian Rhythm; CLOCK Pro | 2016 |
Glucocorticoid receptor haploinsufficiency causes hypertension and attenuates hypothalamic-pituitary-adrenal axis and blood pressure adaptions to high-fat diet.
Topics: Adiposity; Aldosterone; Angiotensins; Animals; Blood Glucose; Blood Pressure; Cell Line; Diet; Dieta | 2008 |
Excess aldosterone-induced changes in insulin signaling molecules and glucose oxidation in gastrocnemius muscle of adult male rat.
Topics: Aldosterone; Animals; Arrestins; beta-Arrestin 2; beta-Arrestins; Blood Glucose; CSK Tyrosine-Protei | 2013 |
Prospective evaluation and characteristics of patients with suspected primary hyperaldosteronism.
Topics: Adult; Aldosterone; Body Mass Index; Female; Glucose Intolerance; Humans; Hyperaldosteronism; Hypert | 2007 |