aldosterone has been researched along with Familial Hypokalemia-Hypomagnesemia in 9 studies
Excerpt | Relevance | Reference |
---|---|---|
"sPRR in patients with primary aldosteronism (low renin-low prorenin) and Gitelman syndrome (high renin-high prorenin) were similar and ≈10% higher than in healthy subjects." | 1.40 | Plasma soluble (pro)renin receptor is independent of plasma renin, prorenin, and aldosterone concentrations but is affected by ethnicity. ( Azizi, M; Baron, S; Bergerot, D; Blanchard, A; Caumont-Prim, A; Chambon, Y; Curis, E; Frank, M; Hirose, T; Nguyen, G; Tabard, SB; Totsune, K, 2014) |
"Overlapping biochemical features in Gitelman's syndrome and Bartter's syndrome has been observed." | 1.36 | Problems in diagnosing atypical Gitelman's syndrome presenting with normomagnesaemia. ( Aoki, K; Koike, T; Kondo, T; Miyoshi, H; Nagai, S; Nakamura, A; Shimizu, C; Tajima, T; Terauchi, Y; Wada, N; Yoshida, M; Yoshioka, N, 2010) |
"Some diseases, such as Gitelman's syndrome, Bartter's syndrome, and primary hyperaldosteronism (Conn's syndrome), may bear some similar clinical and laboratory findings." | 1.35 | Hypokalemic paralysis due to primary hyperaldosteronism simulating Gitelman's syndrome. ( Akalin, A; Cansu, DU; Kasifoglu, T; Korkmaz, C, 2009) |
"As Bartter's and Gitelman's syndrome patients (BS/GS) have increased Ang II, and yet normo/hypotension, hyporesponsiveness to pressors and blunted Ang II signaling via type 1 receptors (AT1R), this study assesses BS/GS's left ventricular (LV) mass and structure as well as Ang II induced ERK1/2 phosphorylation compared with essential hypertensive patients (EH) and normotensive healthy subjects (C) to gain insight into Ang II mediated processes." | 1.35 | High angiotensin II state without cardiac remodeling (Bartter's and Gitelman's syndromes): are angiotensin II type 2 receptors involved? ( Calò, LA; D'Angelo, A; Davis, PA; Montisci, R; Mormino, P; Pagnin, E; Palatini, P; Pessina, AC; Schiavo, S; Scognamiglio, R; Semplicini, A, 2009) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (22.22) | 29.6817 |
2010's | 6 (66.67) | 24.3611 |
2020's | 1 (11.11) | 2.80 |
Authors | Studies |
---|---|
Pathare, G | 1 |
Anderegg, M | 1 |
Albano, G | 1 |
Lang, F | 1 |
Fuster, DG | 1 |
Blanchard, A | 2 |
Brailly Tabard, S | 1 |
Lamaziere, A | 1 |
Bergerot, D | 2 |
Zhygalina, V | 1 |
Lorthioir, A | 1 |
Jacques, A | 1 |
Hourton, D | 1 |
Azizi, M | 2 |
Crambert, G | 1 |
Nguyen, G | 1 |
Curis, E | 1 |
Chambon, Y | 1 |
Hirose, T | 1 |
Caumont-Prim, A | 1 |
Tabard, SB | 1 |
Baron, S | 1 |
Frank, M | 1 |
Totsune, K | 1 |
Kasifoglu, T | 1 |
Akalin, A | 1 |
Cansu, DU | 1 |
Korkmaz, C | 1 |
Nakamura, A | 1 |
Shimizu, C | 1 |
Nagai, S | 1 |
Yoshida, M | 1 |
Aoki, K | 1 |
Kondo, T | 1 |
Miyoshi, H | 1 |
Wada, N | 1 |
Tajima, T | 1 |
Terauchi, Y | 1 |
Yoshioka, N | 1 |
Koike, T | 1 |
Calò, LA | 1 |
Montisci, R | 1 |
Scognamiglio, R | 1 |
Davis, PA | 1 |
Pagnin, E | 1 |
Schiavo, S | 1 |
Mormino, P | 1 |
Semplicini, A | 1 |
Palatini, P | 1 |
D'Angelo, A | 1 |
Pessina, AC | 1 |
Seyberth, HW | 1 |
Schlingmann, KP | 1 |
Walsh, SB | 1 |
Unwin, E | 1 |
Vargas-Poussou, R | 1 |
Houillier, P | 1 |
Unwin, R | 1 |
Balavoine, AS | 1 |
Bataille, P | 1 |
Vanhille, P | 1 |
Azar, R | 1 |
Noël, C | 1 |
Asseman, P | 1 |
Soudan, B | 1 |
Wémeau, JL | 1 |
Vantyghem, MC | 1 |
Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
---|---|---|---|---|---|---|---|
Monocentric STUDY, Randomised Double Blinded (Healthy Subjects, or Transversal (Patients With Gitelman Syndrome)[NCT02297048] | Phase 4 | 26 participants (Actual) | Interventional | 2014-07-31 | Completed | ||
[information is prepared from clinicaltrials.gov, extracted Sep-2024] |
1 review available for aldosterone and Familial Hypokalemia-Hypomagnesemia
Article | Year |
---|---|
Bartter- and Gitelman-like syndromes: salt-losing tubulopathies with loop or DCT defects.
Topics: Adult; Aldosterone; Bartter Syndrome; Female; Gitelman Syndrome; Humans; Hyperaldosteronism; Kidney | 2011 |
8 other studies available for aldosterone and Familial Hypokalemia-Hypomagnesemia
Article | Year |
---|---|
Elevated FGF23 Levels in Mice Lacking the Thiazide-Sensitive NaCl cotransporter (NCC).
Topics: Aldosterone; Analysis of Variance; Animals; Calcium; Disease Models, Animal; Eplerenone; Femur; Fibr | 2018 |
Adrenal adaptation in potassium-depleted men: role of progesterone?
Topics: Adolescent; Adrenal Glands; Adult; Aged; Aldosterone; Animals; Case-Control Studies; Chromatography, | 2020 |
Plasma soluble (pro)renin receptor is independent of plasma renin, prorenin, and aldosterone concentrations but is affected by ethnicity.
Topics: Adolescent; Adult; Aged; Aldosterone; Black People; Diabetes Mellitus, Type 1; Diabetes Mellitus, Ty | 2014 |
Hypokalemic paralysis due to primary hyperaldosteronism simulating Gitelman's syndrome.
Topics: Aldosterone; Biopsy; Diagnosis, Differential; Electromyography; Female; Gitelman Syndrome; Humans; H | 2009 |
Problems in diagnosing atypical Gitelman's syndrome presenting with normomagnesaemia.
Topics: Adolescent; Adult; Aldosterone; DNA Mutational Analysis; Female; Gitelman Syndrome; Humans; Kidney F | 2010 |
High angiotensin II state without cardiac remodeling (Bartter's and Gitelman's syndromes): are angiotensin II type 2 receptors involved?
Topics: Adolescent; Adult; Aldosterone; Analysis of Variance; Angiotensin II; Bartter Syndrome; Blotting, We | 2009 |
Does hypokalaemia cause nephropathy? An observational study of renal function in patients with Bartter or Gitelman syndrome.
Topics: Adult; Aldosterone; Bartter Syndrome; Creatinine; Female; Gitelman Syndrome; Glomerular Filtration R | 2011 |
Phenotype-genotype correlation and follow-up in adult patients with hypokalaemia of renal origin suggesting Gitelman syndrome.
Topics: Adolescent; Adult; Age of Onset; Aged; Aldosterone; Blood Pressure; Body Weight; Child, Preschool; C | 2011 |