aica ribonucleotide has been researched along with Colorectal Neoplasms in 7 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 5 (71.43) | 24.3611 |
2020's | 2 (28.57) | 2.80 |
Authors | Studies |
---|---|
Li, Q; Li, S; Yan, S; Yuan, D; Zhang, F | 1 |
Athineos, D; Blanco, GR; Blyth, K; Brunton, H; Dhayade, S; Fernandez-de-Cossio-Diaz, J; Lilla, S; Mackay, GM; Meiser, J; Oizel, K; Pietzke, M; Sumpton, D; Tait-Mulder, J; Vazquez, A; Zanivan, SR | 1 |
Chang, SF; Chen, CN; Huang, WS; Lee, KC; Lin, CT; Liu, JL | 1 |
Gu, Y; Guo, SY; Huo, HZ; Liu, WY; Qin, J; Wang, B | 1 |
Chen, MB; Han, JY; Li, C; Lu, PH; Shen, W; Wang, J; Wei, MX; Wu, XY | 1 |
Chen, W; Fang, Y; Gong, L; Han, W; He, C; Hu, H; Jin, W; Li, D; Lou, F; Lou, H; Pan, H; Pan, Q; Sui, X; Wang, K; Wang, X; Xu, Y; Yang, J; Zhang, M; Zheng, Y; Zhou, X | 1 |
Kaisani, AA; Kim, SB; Ly, P; Marian, G; Shay, JW; Wright, WE | 1 |
7 other study(ies) available for aica ribonucleotide and Colorectal Neoplasms
Article | Year |
---|---|
AICAR enhances the cytotoxicity of PFKFB3 inhibitor in an AMPK signaling-independent manner in colorectal cancer cells.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Apoptosis; Cell Line, Tumor; Cell Survival; Colorectal Neoplasms; Humans; Phosphofructokinase-2; Ribonucleotides; Signal Transduction | 2021 |
Formate induces a metabolic switch in nucleotide and energy metabolism.
Topics: Adenosine Triphosphate; Adenylate Kinase; Aminoimidazole Carboxamide; Animals; Cell Line, Tumor; Colorectal Neoplasms; Disease Models, Animal; Energy Metabolism; Female; Formates; Humans; Mice, Inbred C57BL; Models, Biological; Models, Genetic; Nucleotides; Orotic Acid; Pyrimidines; Ribonucleotides | 2020 |
Effect of AICAR and 5-Fluorouracil on X-ray Repair, Cross-Complementing Group 1 Expression, and Consequent Cytotoxicity Regulation in Human HCT-116 Colorectal Cancer Cells.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinase Kinases; Antineoplastic Agents; Apoptosis; Cell Proliferation; Cell Survival; Colorectal Neoplasms; Drug Resistance, Neoplasm; Drug Synergism; Drug Therapy, Combination; Fluorouracil; HCT116 Cells; Humans; Protein Kinases; Receptors, CXCR4; Ribonucleotides; X-ray Repair Cross Complementing Protein 1 | 2017 |
AMP-activated protein kinase (AMPK)/Ulk1-dependent autophagic pathway contributes to C6 ceramide-induced cytotoxic effects in cultured colorectal cancer HT-29 cells.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Antineoplastic Agents; Apoptosis; Autophagy; Autophagy-Related Protein-1 Homolog; Cell Survival; Ceramides; Colorectal Neoplasms; Enzyme Activation; Everolimus; HCT116 Cells; HT29 Cells; Humans; Intracellular Signaling Peptides and Proteins; Protein Serine-Threonine Kinases; Ribonucleotides; Signal Transduction; Sirolimus | 2013 |
MicroRNA-451 regulates AMPK/mTORC1 signaling and fascin1 expression in HT-29 colorectal cancer.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Carrier Proteins; Cell Movement; Cell Proliferation; Colorectal Neoplasms; Enzyme Activation; Gene Expression Regulation, Neoplastic; Gene Knockdown Techniques; HEK293 Cells; HT29 Cells; Humans; Mechanistic Target of Rapamycin Complex 1; Microfilament Proteins; MicroRNAs; Models, Biological; Multiprotein Complexes; Ribonucleotides; RNA Interference; Signal Transduction; TOR Serine-Threonine Kinases | 2014 |
Use of metformin alone is not associated with survival outcomes of colorectal cancer cell but AMPK activator AICAR sensitizes anticancer effect of 5-fluorouracil through AMPK activation.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Animals; Antineoplastic Agents; Apoptosis; Autophagy; Cell Cycle Checkpoints; Cell Line, Tumor; Cell Proliferation; Cell Survival; Colorectal Neoplasms; Drug Synergism; Enzyme Activation; Enzyme Activators; Female; Fluorouracil; Humans; Metformin; Mice; Ribonucleotides; Xenograft Model Antitumor Assays | 2014 |
Aneuploid human colonic epithelial cells are sensitive to AICAR-induced growth inhibition through EGFR degradation.
Topics: Aminoimidazole Carboxamide; Aneuploidy; Animals; Antineoplastic Agents; Cell Line, Tumor; Cell Proliferation; Colon; Colorectal Neoplasms; Epithelial Cells; ErbB Receptors; Humans; Hypoglycemic Agents; Intestinal Mucosa; Leupeptins; Mice; Neoplasm Transplantation; Proteasome Endopeptidase Complex; Ribonucleotides; Transplantation, Heterologous; Trisomy; Ubiquitination | 2013 |