aica ribonucleotide has been researched along with Cancer of Nasopharynx in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Dawson, CW; Ko, CW; Lo, AK; Lo, KW; Young, LS | 1 |
Chen, ZT; Guo, SY; Li, L; Liang, ZG; Lu, XD; Qu, S; Su, F; Zhao, W; Zhu, XD | 1 |
2 other study(ies) available for aica ribonucleotide and Cancer of Nasopharynx
Article | Year |
---|---|
Inhibition of the LKB1-AMPK pathway by the Epstein-Barr virus-encoded LMP1 promotes proliferation and transformation of human nasopharyngeal epithelial cells.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinase Kinases; AMP-Activated Protein Kinases; Carcinoma; Cell Line, Tumor; Cell Proliferation; Cell Transformation, Neoplastic; Enzyme Activation; Enzyme Inhibitors; Epithelial Cells; Epstein-Barr Virus Infections; Fluorouracil; Herpesvirus 4, Human; Humans; MAP Kinase Signaling System; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; Nasopharynx; Phosphorylation; Protein Serine-Threonine Kinases; Protein Structure, Tertiary; Ribonucleotides; Signal Transduction; Viral Matrix Proteins | 2013 |
PARP-1 promotes autophagy via the AMPK/mTOR pathway in CNE-2 human nasopharyngeal carcinoma cells following ionizing radiation, while inhibition of autophagy contributes to the radiation sensitization of CNE-2 cells.
Topics: Aminoimidazole Carboxamide; AMP-Activated Protein Kinases; Autophagy; Carcinoma; Cell Line, Tumor; Cell Survival; Humans; Microscopy, Fluorescence; Microtubule-Associated Proteins; Nasopharyngeal Carcinoma; Nasopharyngeal Neoplasms; Phosphorylation; Poly (ADP-Ribose) Polymerase-1; Poly(ADP-ribose) Polymerases; Radiation, Ionizing; Ribonucleotides; Ribosomal Protein S6 Kinases, 70-kDa; RNA Interference; RNA, Small Interfering; Signal Transduction; TOR Serine-Threonine Kinases | 2015 |