Page last updated: 2024-09-03

ah 6809 and Pancreatic Neoplasms

ah 6809 has been researched along with Pancreatic Neoplasms in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Hijikata, T; Ichikawa, H; Morimoto, Y; Takahashi, T; Tsuneyama, K1
Bando, Y; Izumi, K; Ogawa, H; Takahashi, T; Uehara, H; Umemoto, H1
Abruzzese, JL; Cognetti, F; McConkey, DJ; Nawrocki, ST; Pino, MS; Xiong, HQ1

Other Studies

3 other study(ies) available for ah 6809 and Pancreatic Neoplasms

ArticleYear
Inhibition of EP2/EP4 prostanoid receptor-mediated signaling suppresses IGF-1-induced proliferation of pancreatic cancer BxPC-3 cells via upregulating γ-glutamyl cyclotransferase expression.
    Biochemical and biophysical research communications, 2019, 08-20, Volume: 516, Issue:2

    Topics: Cell Line, Tumor; Cell Proliferation; gamma-Aminobutyric Acid; gamma-Glutamylcyclotransferase; Humans; Insulin-Like Growth Factor I; Isoindoles; Metabolome; Pancreatic Neoplasms; Pyrrolidonecarboxylic Acid; Receptors, Prostaglandin E, EP2 Subtype; Receptors, Prostaglandin E, EP4 Subtype; Signal Transduction; Sulfonamides; Up-Regulation; Xanthones

2019
Inhibition of EP2/EP4 signaling abrogates IGF-1R-mediated cancer cell growth: involvement of protein kinase C-θ activation.
    Oncotarget, 2015, Mar-10, Volume: 6, Issue:7

    Topics: Animals; Cell Line, Tumor; Cell Proliferation; Drug Interactions; Enzyme Activation; Humans; Insulin-Like Growth Factor I; Isoindoles; Mice; Mice, Nude; Pancreatic Neoplasms; Protein Kinase C-delta; Receptor Cross-Talk; Receptors, Prostaglandin E, EP2 Subtype; Receptors, Prostaglandin E, EP4 Subtype; Sulfonamides; Xanthones; Xenograft Model Antitumor Assays

2015
Prostaglandin E2 drives cyclooxygenase-2 expression via cyclic AMP response element activation in human pancreatic cancer cells.
    Cancer biology & therapy, 2005, Volume: 4, Issue:11

    Topics: Cell Line, Tumor; Cyclic AMP; Cyclic AMP Response Element-Binding Protein; Cyclic AMP-Dependent Protein Kinases; Cyclooxygenase 2; Dinoprostone; Enzyme Activation; Gene Expression Regulation, Enzymologic; Gene Expression Regulation, Neoplastic; Humans; Isoquinolines; Models, Biological; Pancreatic Neoplasms; Phosphorylation; Prostaglandin Antagonists; Sulfonamides; Xanthones

2005