ag-490 and Hypoxia-Ischemia--Brain

ag-490 has been researched along with Hypoxia-Ischemia--Brain* in 1 studies

Other Studies

1 other study(ies) available for ag-490 and Hypoxia-Ischemia--Brain

Article	Year
Neuroprotective Effects of AG490 in Neonatal Hypoxic-Ischemic Brain Injury. Molecular neurobiology, 2019, Volume: 56, Issue:12 In infants and children, neonatal hypoxic-ischemic (HI) brain injury represents a major cause of chronic neurological morbidity. The transient receptor potential melastatin 2 (TRPM2), a non-selective cation channel that conducts calcium, can mediate neuronal death following HI brain injury. An important endogenous activator of TRPM2 is H Topics: Animals; Animals, Newborn; Cell Survival; Enzyme Inhibitors; HEK293 Cells; Humans; Hypoxia-Ischemia, Brain; Mice; Neuroprotective Agents; Oxidants; Random Allocation; TRPM Cation Channels; Tyrphostins	2019

Article

Year

Neuroprotective Effects of AG490 in Neonatal Hypoxic-Ischemic Brain Injury.

Molecular neurobiology, 2019, Volume: 56, Issue:12

In infants and children, neonatal hypoxic-ischemic (HI) brain injury represents a major cause of chronic neurological morbidity. The transient receptor potential melastatin 2 (TRPM2), a non-selective cation channel that conducts calcium, can mediate neuronal death following HI brain injury. An important endogenous activator of TRPM2 is H

Topics: Animals; Animals, Newborn; Cell Survival; Enzyme Inhibitors; HEK293 Cells; Humans; Hypoxia-Ischemia, Brain; Mice; Neuroprotective Agents; Oxidants; Random Allocation; TRPM Cation Channels; Tyrphostins

2019