adenosine monophosphate has been researched along with Apraxias in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 3 (75.00) | 29.6817 |
| 2010's | 1 (25.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kunkel, TA; Schellenberg, MJ; Tumbale, P; Williams, JS; Williams, RS | 1 |
| Caldecott, KW; El-Khamisy, SF; Reynolds, JJ | 1 |
| Bieganowski, P; Brenner, C; Seidle, HF | 1 |
| Date, H; Igarashi, S; Koyama, A; Nishizawa, M; Onodera, O; Shiga, A; Tada, M; Takahashi, T; Tsuji, S; Yokoseki, A; Yoshida, Y | 1 |
1 review(s) available for adenosine monophosphate and Apraxias
| Article | Year |
|---|---|
Short-patch single-strand break repair in ataxia oculomotor apraxia-1.
Topics: Adenosine Monophosphate; Animals; Apraxias; Ataxia; DNA Breaks, Single-Stranded; DNA Repair; DNA-Binding Proteins; Humans; Mutation; Nuclear Proteins | 2009 |
3 other study(ies) available for adenosine monophosphate and Apraxias
| Article | Year |
|---|---|
Aprataxin resolves adenylated RNA-DNA junctions to maintain genome integrity.
Topics: Adenosine Monophosphate; Apraxias; Ataxia Telangiectasia; Cell Survival; Cerebellar Ataxia; DNA; DNA Repair; DNA-Binding Proteins; Exodeoxyribonucleases; Genome, Human; Humans; Hypoalbuminemia; Models, Molecular; Mutation; Nuclear Proteins; Nucleic Acid Conformation; Protein Conformation; Protein Folding; Ribonuclease H; RNA; S Phase Cell Cycle Checkpoints; Saccharomyces cerevisiae; Schizosaccharomyces; Schizosaccharomyces pombe Proteins; Structure-Activity Relationship; Zinc | 2014 |
Disease-associated mutations inactivate AMP-lysine hydrolase activity of Aprataxin.
Topics: Adenosine Monophosphate; Alleles; Apraxias; Ataxia; Binding Sites; Blotting, Western; Cations; DNA Damage; DNA-Binding Proteins; Electrophoresis, Polyacrylamide Gel; Escherichia coli; Eye Diseases; Humans; Hydrogen-Ion Concentration; Hydrolases; Kinetics; Lysine; Metals; Motor Neuron Disease; Mutagenesis, Site-Directed; Mutation; Nuclear Proteins; Phenotype; Plasmids; Protein Binding; Protein Structure, Tertiary; RNA, Messenger; Substrate Specificity; Syndrome; Temperature; Zinc Fingers | 2005 |
Aprataxin, causative gene product for EAOH/AOA1, repairs DNA single-strand breaks with damaged 3'-phosphate and 3'-phosphoglycolate ends.
Topics: Adenosine Monophosphate; Apraxias; Ataxia; DNA; DNA Breaks, Single-Stranded; DNA Ligase ATP; DNA Ligases; DNA Polymerase beta; DNA Repair; DNA-Binding Proteins; Glycolates; Guanosine Monophosphate; Humans; Mutation; Nuclear Proteins; Phosphates; Phosphoric Monoester Hydrolases; Poly-ADP-Ribose Binding Proteins; Protein Structure, Tertiary; Substrate Specificity; Xenopus Proteins | 2007 |