adenosine diphosphate has been researched along with Colon Cancer, Familial Nonpolyposis in 3 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 2 (66.67) | 29.6817 |
2010's | 1 (33.33) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Cyr, JL; Heinen, CD | 1 |
Cook, C; Cyr, JL; Fishel, R; Forties, RA; Heinen, CD; Hingorani, MM; Lopez, JM; Punja, N; Sakato, M | 1 |
Berardini, M; Butz, C; Fishel, R; Heinen, CD; Mazurek, A; Wilson, T | 1 |
3 other study(ies) available for adenosine diphosphate and Colon Cancer, Familial Nonpolyposis
Article | Year |
---|---|
Hereditary cancer-associated missense mutations in hMSH6 uncouple ATP hydrolysis from DNA mismatch binding.
Topics: Adenosine Diphosphate; Adenosine Triphosphatases; Adenosine Triphosphate; Base Pair Mismatch; Colorectal Neoplasms, Hereditary Nonpolyposis; DNA-Binding Proteins; Humans; Hydrolysis; Models, Molecular; Mutation, Missense; MutS Homolog 2 Protein; Protein Binding; Protein Structure, Quaternary; Protein Structure, Tertiary | 2008 |
Human MSH2 (hMSH2) protein controls ATP processing by hMSH2-hMSH6.
Topics: Adenosine Diphosphate; Adenosine Triphosphate; Colorectal Neoplasms, Hereditary Nonpolyposis; DNA-Binding Proteins; Humans; Hydrolysis; Kinetics; Multienzyme Complexes; MutS Homolog 2 Protein | 2011 |
HNPCC mutations in hMSH2 result in reduced hMSH2-hMSH6 molecular switch functions.
Topics: Adenosine Diphosphate; Adenosine Triphosphatases; Adenosine Triphosphate; Base Pair Mismatch; Binding Sites; Colorectal Neoplasms, Hereditary Nonpolyposis; Dimerization; DNA; DNA Repair; DNA-Binding Proteins; Electrophoretic Mobility Shift Assay; Humans; Hydrolysis; Mutagenesis; Mutation, Missense; MutS Homolog 2 Protein; Protein Binding; Proto-Oncogene Proteins | 2002 |