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adenosine diphosphate and Cardiac Remodeling, Ventricular

adenosine diphosphate has been researched along with Cardiac Remodeling, Ventricular in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's2 (40.00)29.6817
2010's2 (40.00)24.3611
2020's1 (20.00)2.80

Authors

AuthorsStudies
Agrimi, J; Bottomley, PA; Dey, S; Gabr, R; Gerstenblith, G; Gupta, A; Keceli, G; Lai, S; Leppo, M; O'Rourke, B; Paolocci, N; Schär, M; Sourdon, J; Steenbergen, C; Stuber, A; Tocchetti, CG; Wang, Y; Weiss, RG; Yanek, LR; Zhang, Y1
de Wijs-Meijler, D; Duncker, DJ; Jan Danser, AH; Jankowski, J; Jankowski, V; Lankhuizen, I; Merkus, D; Zhou, Z1
Chen, A; Chen, M; Chen, X; Dai, W; Dong, Q; Li, W; Li, X; Ou, C; Shen, Y1
Bache, RJ; Bransford, TL; Feygin, J; From, AH; Hu, Q; Lee, J; Liu, J; Mansoor, A; Ochiai, K; Swingen, C; Wang, X; Zeng, L; Zhang, G; Zhang, J1
Bache, RJ; Godinot, C; Gong, G; Ishibashi, Y; Liu, J; Murakami, Y; Ochiai, K; Prody, C; Wang, C; Zhang, J1

Other Studies

5 other study(ies) available for adenosine diphosphate and Cardiac Remodeling, Ventricular

ArticleYear
Mitochondrial Creatine Kinase Attenuates Pathologic Remodeling in Heart Failure.
    Circulation research, 2022, 03-04, Volume: 130, Issue:5

    Topics: Adenosine Diphosphate; Adenosine Triphosphate; Animals; Creatine Kinase; Creatine Kinase, Mitochondrial Form; Energy Metabolism; Heart Failure; Humans; Hypertrophy, Left Ventricular; Mice; Myocardium; Reactive Oxygen Species; Ventricular Remodeling

2022
Blunted coronary vasodilator response to uridine adenosine tetraphosphate in post-infarct remodeled myocardium is due to reduced P1 receptor activation.
    Pharmacological research, 2013, Volume: 77

    Topics: Adenosine A2 Receptor Antagonists; Adenosine Diphosphate; Animals; Coronary Vessels; Dinucleoside Phosphates; Heart; Myocardial Infarction; Myocardium; Purinergic P1 Receptor Agonists; Purinergic P1 Receptor Antagonists; Purinergic P2Y Receptor Antagonists; Pyridoxal Phosphate; Pyrimidines; Receptors, Purinergic; Receptors, Purinergic P1; Theophylline; Triazoles; Vasodilator Agents; Ventricular Remodeling

2013
Trimetazidine attenuates pressure overload-induced early cardiac energy dysfunction via regulation of neuropeptide Y system in a rat model of abdominal aortic constriction.
    BMC cardiovascular disorders, 2016, 11-17, Volume: 16, Issue:1

    Topics: Adenosine Diphosphate; Adenosine Triphosphate; Animals; Aorta, Abdominal; Arterial Pressure; Cardiovascular Agents; Constriction; Disease Models, Animal; Energy Metabolism; Gene Expression Regulation; Hypertrophy, Left Ventricular; Male; Myocardium; Neuropeptide Y; Oxidative Stress; Rats, Wistar; Receptors, G-Protein-Coupled; Receptors, Neuropeptide; Receptors, Neuropeptide Y; Signal Transduction; Trimetazidine; Ventricular Function, Left; Ventricular Remodeling

2016
Profound bioenergetic abnormalities in peri-infarct myocardial regions.
    American journal of physiology. Heart and circulatory physiology, 2006, Volume: 291, Issue:2

    Topics: Adenosine Diphosphate; Adenosine Triphosphatases; Adenosine Triphosphate; Animals; Blotting, Western; Citrate (si)-Synthase; Collagen; Coronary Circulation; Coronary Vessels; Energy Metabolism; Hemodynamics; Ligation; Magnetic Resonance Spectroscopy; Mitochondria, Heart; Myocardial Infarction; Myocardium; Myoglobin; Oxygen Consumption; Phosphocreatine; Swine; Ventricular Remodeling

2006
Mitochondrial ATPase and high-energy phosphates in failing hearts.
    American journal of physiology. Heart and circulatory physiology, 2001, Volume: 281, Issue:3

    Topics: Adenosine Diphosphate; Adenosine Triphosphate; Animals; Body Weight; Cardiac Pacing, Artificial; Coronary Circulation; Disease Models, Animal; Dobutamine; Dopamine; Heart Failure; Hemodynamics; Infusions, Intravenous; Magnetic Resonance Spectroscopy; Mitochondria, Heart; Myocardial Infarction; Organ Size; Phosphocreatine; Protein Subunits; Proton-Translocating ATPases; Swine; Ventricular Remodeling

2001