activins and Wasting Disease

activins has been researched along with Wasting Disease in 9 studies

Research

Studies (9)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's1 (11.11)18.2507
2000's5 (55.56)29.6817
2010's2 (22.22)24.3611
2020's1 (11.11)2.80

Authors

AuthorsStudies
Boor, P; Cohen, CD; Eichinger, F; Hoxha, E; Huber, TB; Kilic, T; Klinkhammer, BM; Kretz, O; Kretzler, M; Kylies, D; Lindenmeyer, MT; Liu, S; Lu, S; Martin, IV; Matsakas, A; Menon, R; Meyer, C; Mitchell, R; Nair, V; Omairi, S; Ostendorf, T; Paolini, A; Pasternack, A; Patel, K; Puelles, VG; Ritvos, O; Sandri, M; Solagna, F; Tezze, C; Tharaux, PL; Turner, JE; Wanner, N; Wiesch, JSZ; Wu, G; Zhao, Y1
Brebbia, P; Geisse, S; Glass, DJ; Jacobi, C; Koch, E; Koelbing, C; Lach-Trifilieff, E; Leber, X; Lehmann, S; Minetti, G; Morvan, F; Ritter, V; Rondeau, JM; Scharenberg, M; Scheufler, C; Schilb, A; Toussaint, G; Witte, F; Zou, C1
Bertona, M; Emanuele, E; Fiuza-Luces, C; Lucia, A; Morales, JS; Pareja-Galeano, H; Sanchis-Gomar, F1
Brown, CA; Chen, L; Matzuk, MM; Wu, X; Yan, C1
Graff, JM; Li, Q; Loveland, KL; Matzuk, MM; O'Connor, AE1
Kumar, R; Li, Q; Loveland, KL; Matzuk, MM; O'Connor, AE; Seehra, JS; Underwood, K1
Boime, I; Byrne, MC; Kumar, TR; Matzuk, MM; Palapattu, G; Wang, P; Woodruff, TK1
Chen, L; Cipriano, SC; Kumar, TR; Matzuk, MM1
Davies, MV; Esquela, AF; Haynes, P; Koniaris, LG; Lee, SJ; McPherron, AC; Tomkinson, KN; Wolfman, NM; Zimmers, TA1

Other Studies

9 other study(ies) available for activins and Wasting Disease

ArticleYear
Pro-cachectic factors link experimental and human chronic kidney disease to skeletal muscle wasting programs.
    The Journal of clinical investigation, 2021, 06-01, Volume: 131, Issue:11

    Topics: Activin Receptors, Type II; Activins; Animals; Cachexia; Disease Models, Animal; HEK293 Cells; Humans; Mice; Mice, Knockout; Muscle, Skeletal; Muscular Atrophy; Renal Insufficiency, Chronic; Wasting Syndrome

2021
Blockade of activin type II receptors with a dual anti-ActRIIA/IIB antibody is critical to promote maximal skeletal muscle hypertrophy.
    Proceedings of the National Academy of Sciences of the United States of America, 2017, 11-21, Volume: 114, Issue:47

    Topics: Activin Receptors, Type II; Activins; Animals; Antibodies, Blocking; Antibodies, Monoclonal; Antibodies, Monoclonal, Humanized; Bone Morphogenetic Proteins; Crystallography, X-Ray; Dose-Response Relationship, Drug; Growth Differentiation Factors; HEK293 Cells; Humans; Hypertrophy; Male; Mice; Mice, SCID; Muscle, Skeletal; Myostatin; Rats; Rats, Wistar; Recombinant Proteins; Signal Transduction; Wasting Syndrome

2017
Baicalin supplementation reduces serum biomarkers of skeletal muscle wasting and may protect against lean body mass reduction in cancer patients: Results from a pilot open-label study.
    Neuro endocrinology letters, 2016, Volume: 37, Issue:3

    Topics: Absorptiometry, Photon; Activins; Aged; Biomarkers; Body Weight; Cachexia; Endpoint Determination; Female; Flavonoids; Humans; Male; Middle Aged; Muscle, Skeletal; Muscular Atrophy; Neoplasms; NF-kappa B; Pilot Projects; Wasting Syndrome; Weight Loss

2016
Interrelationship of growth differentiation factor 9 and inhibin in early folliculogenesis and ovarian tumorigenesis in mice.
    Molecular endocrinology (Baltimore, Md.), 2004, Volume: 18, Issue:6

    Topics: Activins; Animals; Bone Morphogenetic Protein 15; Cachexia; Cell Differentiation; Cell Proliferation; DNA-Binding Proteins; Female; Granulosa Cells; Growth Differentiation Factor 9; Homeodomain Proteins; In Situ Hybridization; Inhibins; Intercellular Signaling Peptides and Proteins; Mice; Mice, Knockout; Mice, Transgenic; Oncogene Proteins; Ovarian Follicle; Ovarian Neoplasms; Protein Structure, Tertiary; Proto-Oncogene Proteins c-kit; Receptors, Cytoplasmic and Nuclear; Reverse Transcriptase Polymerase Chain Reaction; RNA; RNA, Messenger; Stem Cell Factor; Steroidogenic Factor 1; Theca Cells; Time Factors; Transcription Factors; Up-Regulation; Wasting Syndrome

2004
SMAD3 regulates gonadal tumorigenesis.
    Molecular endocrinology (Baltimore, Md.), 2007, Volume: 21, Issue:10

    Topics: Activins; Animals; Cell Transformation, Neoplastic; Female; Follicle Stimulating Hormone; Inhibins; Male; Mice; Mice, Knockout; Ovarian Neoplasms; RNA, Messenger; Smad3 Protein; Testicular Neoplasms; Wasting Syndrome; Weight Loss

2007
Prevention of cachexia-like syndrome development and reduction of tumor progression in inhibin-deficient mice following administration of a chimeric activin receptor type II-murine Fc protein.
    Molecular human reproduction, 2007, Volume: 13, Issue:9

    Topics: Activin Receptors, Type II; Activins; Animals; Cachexia; Disease Progression; Female; Follicle Stimulating Hormone; Gastric Mucosa; Immunoglobulin Fc Fragments; Immunoglobulin G; Inhibins; Liver; Male; Mice; Mice, Knockout; Recombinant Fusion Proteins; Sex Cord-Gonadal Stromal Tumors; Stomach; Syndrome; Testicular Neoplasms; Wasting Syndrome

2007
Transgenic models to study gonadotropin function: the role of follicle-stimulating hormone in gonadal growth and tumorigenesis.
    Molecular endocrinology (Baltimore, Md.), 1999, Volume: 13, Issue:6

    Topics: Activins; Animals; Crosses, Genetic; Female; Follicle Stimulating Hormone; Gene Expression Regulation; Hemorrhage; Homozygote; Humans; Infertility, Female; Infertility, Male; Inhibins; Male; Metallothionein; Mice; Mice, Mutant Strains; Mice, Transgenic; Oligopeptides; Ovarian Neoplasms; Ovary; Peptides; Polycystic Ovary Syndrome; Seminal Vesicles; Steroids; Testicular Neoplasms; Urinary Tract; Wasting Syndrome

1999
Follistatin is a modulator of gonadal tumor progression and the activin-induced wasting syndrome in inhibin-deficient mice.
    Endocrinology, 2000, Volume: 141, Issue:7

    Topics: Activins; Animals; Disease Progression; Female; Follistatin; Glycoproteins; Inhibins; Liver Diseases; Male; Metallothionein; Mice; Mice, Transgenic; Ovarian Neoplasms; Peptides; Stomach Diseases; Survival Analysis; Testicular Neoplasms; Wasting Syndrome

2000
Induction of cachexia in mice by systemically administered myostatin.
    Science (New York, N.Y.), 2002, May-24, Volume: 296, Issue:5572

    Topics: 3T3 Cells; Activins; Adipose Tissue; Animals; Body Weight; Cachexia; CHO Cells; Cricetinae; Eating; Female; Follistatin; Liver; Mice; Mice, Nude; Muscle Fibers, Skeletal; Muscle, Skeletal; Myostatin; Organ Size; Peptide Fragments; Recombinant Proteins; Transforming Growth Factor beta; Wasting Syndrome; Weight Loss

2002