Page last updated: 2024-10-31

activins and Esophageal Reflux

activins has been researched along with Esophageal Reflux in 1 studies

Activins: Activins are produced in the pituitary, gonads, and other tissues. By acting locally, they stimulate pituitary FSH secretion and have diverse effects on cell differentiation and embryonic development. Activins are glycoproteins that are hetero- or homodimers of INHIBIN-BETA SUBUNITS.

Research Excerpts

ExcerptRelevanceReference
"We identified a novel player in gastroesophageal reflux injury, the TGFβ family member Activin A (ActA), which is a known regulator of inflammation and tissue repair."1.51Activin A-mediated epithelial de-differentiation contributes to injury repair in an in vitro gastrointestinal reflux model. ( Andl, CD; Andl, T; Catala-Valentin, A; Le Bras, GF; Roudebush, C, 2019)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Roudebush, C1
Catala-Valentin, A1
Andl, T1
Le Bras, GF1
Andl, CD1

Other Studies

1 other study available for activins and Esophageal Reflux

ArticleYear
Activin A-mediated epithelial de-differentiation contributes to injury repair in an in vitro gastrointestinal reflux model.
    Cytokine, 2019, Volume: 123

    Topics: Activins; Cell Dedifferentiation; Epithelial Cells; Esophagus; Gastroesophageal Reflux; Humans; Mode

2019