activins has been researched along with Cancer of Mouth in 7 studies
Activins: Activins are produced in the pituitary, gonads, and other tissues. By acting locally, they stimulate pituitary FSH secretion and have diverse effects on cell differentiation and embryonic development. Activins are glycoproteins that are hetero- or homodimers of INHIBIN-BETA SUBUNITS.
| Excerpt | Relevance | Reference |
|---|---|---|
| " Using an orally bioavailable inhibitor of the Activin A pathway to attenuate oral cancer cell migration and invasion, we further demonstrate the targetability of this signaling axis." | 1.91 | ΔNp63 overexpression promotes oral cancer cell migration through hyperactivated Activin A signaling. ( Basu, S; Dalal, S; Kumar, S; Mishra, R; Mundhe, D; Teni, T, 2023) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 4 (57.14) | 24.3611 |
| 2020's | 3 (42.86) | 2.80 |
| Authors | Studies |
|---|---|
| Mundhe, D | 2 |
| Mishra, R | 2 |
| Basu, S | 1 |
| Dalal, S | 1 |
| Kumar, S | 1 |
| Teni, T | 2 |
| Ervolino De Oliveira, C | 1 |
| Dourado, MR | 1 |
| Sawazaki-Calone, Í | 1 |
| Costa De Medeiros, M | 1 |
| Rossa Júnior, C | 1 |
| De Karla Cervigne, N | 1 |
| Esquiche León, J | 1 |
| Lambert, D | 1 |
| Salo, T | 2 |
| Graner, E | 3 |
| Coletta, RD | 3 |
| Waghole, R | 1 |
| Pawar, S | 1 |
| Shetty, A | 1 |
| Gera, P | 1 |
| Kannan, S | 1 |
| Tsai, CN | 2 |
| Tsai, CL | 2 |
| Yi, JS | 1 |
| Kao, HK | 2 |
| Huang, Y | 1 |
| Wang, CI | 1 |
| Lee, YS | 2 |
| Chang, KP | 2 |
| Bufalino, A | 2 |
| Cervigne, NK | 1 |
| de Oliveira, CE | 1 |
| Fonseca, FP | 1 |
| Rodrigues, PC | 1 |
| Macedo, CC | 1 |
| Sobral, LM | 2 |
| Miguel, MC | 1 |
| Lopes, MA | 2 |
| Paes Leme, AF | 1 |
| Lambert, DW | 1 |
| Salo, TA | 1 |
| Kowalski, LP | 1 |
| Liang, Y | 1 |
| Cheng, MH | 1 |
| Chang, YL | 1 |
| Liu, SC | 1 |
| Lin, YC | 1 |
| Ko, TY | 1 |
| Wang, TH | 1 |
| Hao, SP | 1 |
7 other studies available for activins and Cancer of Mouth
| Article | Year |
|---|---|
ΔNp63 overexpression promotes oral cancer cell migration through hyperactivated Activin A signaling.
Topics: Activins; Cell Line, Tumor; Cell Movement; Gene Knockdown Techniques; Humans; Mouth Neoplasms; Signa | 2023 |
Activin A triggers angiogenesis via regulation of VEGFA and its overexpression is associated with poor prognosis of oral squamous cell carcinoma.
Topics: Activins; Adult; Aged; Aged, 80 and over; Autocrine Communication; Cell Movement; Cell Proliferation | 2020 |
Concomitant overexpression of Activin A and p63 is associated with poor outcome in oral cancer patients.
Topics: Activins; Humans; Inhibin-beta Subunits; Membrane Proteins; Mouth Neoplasms | 2020 |
Activin A regulates the epidermal growth factor receptor promoter by activating the PI3K/SP1 pathway in oral squamous cell carcinoma cells.
Topics: Activins; Adult; Aged; Aged, 80 and over; Carcinoma, Squamous Cell; Cell Movement; ErbB Receptors; F | 2019 |
Low miR-143/miR-145 Cluster Levels Induce Activin A Overexpression in Oral Squamous Cell Carcinomas, Which Contributes to Poor Prognosis.
Topics: Activins; Adult; Aged; Apoptosis; Carcinoma, Squamous Cell; Cell Survival; Databases, Genetic; Disea | 2015 |
Overexpression of activin A in oral squamous cell carcinoma: association with poor prognosis and tumor progression.
Topics: Activins; Adult; Aged; Blotting, Western; Carcinoma, Squamous Cell; Cell Adhesion; Cell Movement; Di | 2010 |
Myofibroblasts in the stroma of oral cancer promote tumorigenesis via secretion of activin A.
Topics: Activins; Animals; Carcinoma, Squamous Cell; Cell Proliferation; Down-Regulation; Enzyme-Linked Immu | 2011 |