acetylleucyl-leucyl-norleucinal has been researched along with Agyria in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (50.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Hirotsune, S; Kengaku, M; Miyakawa, T; Mori, D; Sato, M; Sorimachi, H; Takao, K; Takitoh, T; Umeshima, H; Wynshaw-Boris, A; Yamada, M; Yoshida, Y | 1 |
| Baraban, SC; Bershteyn, M; Hirotsune, S; Sebe, JY; Wynshaw-Boris, A | 1 |
2 other study(ies) available for acetylleucyl-leucyl-norleucinal and Agyria
| Article | Year |
|---|---|
Inhibition of calpain increases LIS1 expression and partially rescues in vivo phenotypes in a mouse model of lissencephaly.
Topics: 1-Alkyl-2-acetylglycerophosphocholine Esterase; Animals; Calpain; Cell Movement; Cells, Cultured; Cerebral Cortex; Cysteine Proteinase Inhibitors; Disease Models, Animal; Dyneins; Embryo, Mammalian; Female; Fibroblasts; Gene Expression Regulation, Developmental; Leucine; Leupeptins; Lissencephaly; Mice; Mice, Knockout; Microtubule-Associated Proteins; Models, Neurological; Neurons; Phenotype; Pregnancy | 2009 |
ALLN rescues an in vitro excitatory synaptic transmission deficit in Lis1 mutant mice.
Topics: 1-Alkyl-2-acetylglycerophosphocholine Esterase; Animals; Calpain; Cysteine Proteinase Inhibitors; Excitatory Postsynaptic Potentials; Gene Expression; Heterozygote; Leupeptins; Lissencephaly; Mice; Mice, Mutant Strains; Microtubule-Associated Proteins; Miniature Postsynaptic Potentials; Mutation; Proteolysis; Pyramidal Cells; Spectrin | 2013 |