acetylglucosamine has been researched along with Myocardial Ischemia in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 5 (83.33) | 29.6817 |
| 2010's | 1 (16.67) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Liu, S; Paschen, W; Sheng, H; Yang, W; Yu, Z | 1 |
| Jones, SP; Ngoh, GA | 1 |
| Dillmann, W; Facundo, HT; Hamid, T; Jones, SP; Ngoh, GA; Zachara, NE | 1 |
| Cohen, MV; Downey, JM | 1 |
| Bounelis, P; Chang, T; Chatham, JC; Liu, J; Marchase, RB; Pang, Y | 1 |
| Chatham, JC; Liu, J; Marchase, RB | 1 |
1 review(s) available for acetylglucosamine and Myocardial Ischemia
| Article | Year |
|---|---|
New insights into metabolic signaling and cell survival: the role of beta-O-linkage of N-acetylglucosamine.
Topics: Acetylglucosamine; Animals; Cardiovascular System; Cell Survival; Humans; Metabolism; Myocardial Ischemia; Signal Transduction | 2008 |
5 other study(ies) available for acetylglucosamine and Myocardial Ischemia
| Article | Year |
|---|---|
O-linked β-N-acetylglucosamine modification of proteins is activated in post-ischemic brains of young but not aged mice: Implications for impaired functional recovery from ischemic stress.
Topics: Acetylglucosamine; Aging; Animals; Brain; Brain Ischemia; Male; Mice; Mice, Inbred C57BL; Myocardial Ischemia; Protein Processing, Post-Translational; Psychomotor Performance; Recovery of Function; Stress, Physiological; Stroke; SUMO-1 Protein; Ubiquitination; Unfolded Protein Response | 2016 |
Unique hexosaminidase reduces metabolic survival signal and sensitizes cardiac myocytes to hypoxia/reoxygenation injury.
Topics: Acetylglucosamine; Animals; Animals, Newborn; beta-N-Acetylhexosaminidases; Calcium; Cardiotonic Agents; Cell Hypoxia; Cell Survival; Cells, Cultured; Glycosylation; Ischemic Preconditioning, Myocardial; Membrane Potential, Mitochondrial; Mice; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Ischemia; Myocytes, Cardiac; Oximes; Phenylcarbamates; Protein Biosynthesis; Protein Processing, Post-Translational; Rats; Rats, Sprague-Dawley; Recombinant Fusion Proteins; RNA Interference; RNA, Small Interfering | 2009 |
O-linked beta-N-acetylglucosamine: a new piece of the cardioprotection puzzle?
Topics: Acetylglucosamine; Animals; beta-N-Acetylhexosaminidases; Carbohydrate Conformation; Cardiotonic Agents; Cells, Cultured; Glycosylation; Humans; Ischemic Preconditioning, Myocardial; Mice; Mitochondrial Membrane Transport Proteins; Mitochondrial Permeability Transition Pore; Myocardial Ischemia; Myocytes, Cardiac; N-Acetylglucosaminyltransferases; Oximes; Phenylcarbamates; Protein Processing, Post-Translational; Rats; Voltage-Dependent Anion Channels | 2009 |
Increased hexosamine biosynthesis and protein O-GlcNAc levels associated with myocardial protection against calcium paradox and ischemia.
Topics: Acetylglucosamine; Animals; Calcium; Glycoproteins; Hexosamines; Male; Myocardial Ischemia; Myocardium; Rats; Rats, Sprague-Dawley | 2006 |
Increased O-GlcNAc levels during reperfusion lead to improved functional recovery and reduced calpain proteolysis.
Topics: Acetylglucosamine; Adenosine Triphosphate; Animals; Calcium; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases; Calpain; Carrier Proteins; Glucosamine; Microfilament Proteins; Myocardial Ischemia; Myocardial Reperfusion Injury; Myocardium; Oximes; Phenylcarbamates; Rats | 2007 |