acetylglucosamine has been researched along with Carcinoma, Pancreatic Ductal in 5 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (60.00) | 24.3611 |
| 2020's | 2 (40.00) | 2.80 |
| Authors | Studies |
|---|---|
| Fujii, C; Harumiya, S; Kawakubo, M; Matoba, H; Nakayama, J; Yamanoi, K; Yuki, A | 1 |
| Fan, Z; Geng, D; Lai, Z; Qin, W; Wang, Y; Wu, L; Yang, W; Yi, W; Zhang, J; Zhou, H; Zhou, R; Zhu, Q | 1 |
| Fujii, C; Nakayama, J; Ohya, A; Shimojo, H; Yamanoi, K | 1 |
| Chang, MC; Chang, YT; Chen, YJ; Hsieh, PK; Hu, CM; Jeng, YM; Lee, EYP; Lee, WH; Tien, SC | 1 |
| Allen, PJ; Brand, RE; Cao, Z; Dai, J; Feng, Z; Haab, BB; Hostetter, G; Partyka, K; Simeone, DM; Sinha, J; Tang, H | 1 |
5 other study(ies) available for acetylglucosamine and Carcinoma, Pancreatic Ductal
| Article | Year |
|---|---|
Glycosylation of MUC6 by α1,4-linked N-acetylglucosamine enhances suppression of pancreatic cancer malignancy.
Topics: Acetylglucosamine; Biomarkers, Tumor; Carcinoma, Pancreatic Ductal; Cell Line, Tumor; Cell Movement; Cell Proliferation; Glycosylation; Humans; Mucin-6; N-Acetylglucosaminyltransferases; Pancreatic Neoplasms; Prognosis; RNA, Messenger; Trefoil Factor-2; Tumor Suppressor Proteins | 2022 |
O-GlcNAcylation promotes pancreatic tumor growth by regulating malate dehydrogenase 1.
Topics: Acetylglucosamine; Animals; Carcinoma, Pancreatic Ductal; Cell Line, Tumor; Glutamine; Malate Dehydrogenase; Mice; Mice, Nude; N-Acetylglucosaminyltransferases; Pancreatic Neoplasms; Proto-Oncogene Proteins p21(ras); Serine | 2022 |
Gastric gland mucin-specific O-glycan expression decreases with tumor progression from precursor lesions to pancreatic cancer.
Topics: Acetylglucosamine; Biomarkers, Tumor; Carbohydrate Conformation; Carcinogenesis; Carcinoma, Pancreatic Ductal; Disease Progression; Gastric Mucosa; Glycosylation; Humans; Mucin 5AC; Mucin-6; Pancreatic Neoplasms; Precancerous Conditions; Protein Processing, Post-Translational | 2017 |
High Glucose Triggers Nucleotide Imbalance through O-GlcNAcylation of Key Enzymes and Induces KRAS Mutation in Pancreatic Cells.
Topics: Acetylation; Acetylglucosamine; Acetyltransferases; Adult; Aged; Animals; Carcinoma, Pancreatic Ductal; Cell Transformation, Neoplastic; Cells, Cultured; DNA Damage; Dose-Response Relationship, Drug; Enzymes; Female; Glucose; HEK293 Cells; Humans; Infant, Newborn; Male; Metabolic Networks and Pathways; Mice; Mice, Inbred C57BL; Middle Aged; Mutagenesis; Mutation; Nucleotides; Pancreas; Pancreatic Neoplasms; Protein Processing, Post-Translational; Proto-Oncogene Proteins p21(ras); Young Adult | 2019 |
A Gastric Glycoform of MUC5AC Is a Biomarker of Mucinous Cysts of the Pancreas.
Topics: Acetylglucosamine; Adenocarcinoma, Mucinous; Biomarkers; Carcinoma, Pancreatic Ductal; Carcinoma, Papillary; Cohort Studies; Glycosylation; Heparan Sulfate Proteoglycans; Humans; Mucin 5AC; N-Acetylglucosaminyltransferases; Pancreatic Cyst; Pancreatic Neoplasms; Peptide Fragments | 2016 |