acetylglucosamine has been researched along with Carcinogenesis in 9 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 6 (66.67) | 24.3611 |
2020's | 3 (33.33) | 2.80 |
Authors | Studies |
---|---|
Shibuya, H; Shimizu, M; Tanaka, N | 1 |
Ahmad, M; Dai, Y; Dong, MQ; Li, J; Liu, Y; Lu, M; Mi, W; Sang, L; Sun, J; Wang, X; Wang, Y; Yan, Y; Yang, YG; Zhan, Y; Zhang, R | 1 |
Chen, YY; Khoo, KH; Liu, TW; Parker, JM; Sinclair, DAR; Verheyen, EM; Vocadlo, DJ; Wong, KKL | 1 |
Chen, Y; Liu, X; Pan, Q; Qiao, Y; Sun, F; Wang, J; Wu, Q; Yu, Y; Zhang, X; Zhao, Y; Zhu, G; Zou, S | 1 |
Fujii, C; Nakayama, J; Ohya, A; Shimojo, H; Yamanoi, K | 1 |
Hagan, CR; Trinca, GM | 1 |
Dai, Y; Fan, X; Gao, S; Lin, Y; Liu, X; Liu, Y; Miao, Y; Qian, P; Xia, L; Zhou, J; Zhu, J; Zhu, P | 1 |
Chu, S; Fang, X; Fei, B; Liu, Z; Yu, M | 1 |
Ma, Z; Vosseller, K | 1 |
2 review(s) available for acetylglucosamine and Carcinogenesis
Article | Year |
---|---|
O-GlcNAcylation in women's cancers: breast, endometrial and ovarian.
Topics: Acetylglucosamine; Breast Neoplasms; Carcinogenesis; Endometrial Neoplasms; Female; Glycosylation; Humans; Neoplasms; Ovarian Neoplasms; Protein Processing, Post-Translational | 2018 |
Cancer metabolism and elevated O-GlcNAc in oncogenic signaling.
Topics: Acetylglucosamine; Animals; Carcinogenesis; Epigenesis, Genetic; Humans; Neoplasms; Neovascularization, Pathologic; Signal Transduction | 2014 |
7 other study(ies) available for acetylglucosamine and Carcinogenesis
Article | Year |
---|---|
Enhanced O-GlcNAc modification induced by the RAS/MAPK/CDK1 pathway is required for SOX2 protein expression and generation of cancer stem cells.
Topics: Acetylglucosamine; Animals; Antineoplastic Agents; Carcinogenesis; CDC2 Protein Kinase; Cyclic N-Oxides; Enzyme Inhibitors; Gene Expression; Humans; Indolizines; Mice; Mitogen-Activated Protein Kinases; Neoplastic Stem Cells; Pyridinium Compounds; ras Proteins; Signal Transduction; SOXB1 Transcription Factors | 2022 |
O-GlcNAcylation promotes the cytosolic localization of the m
Topics: Acetylglucosamine; Animals; Carcinogenesis; Colorectal Neoplasms; Humans; Mice; N-Acetylglucosaminyltransferases; Phosphorylation; Protein Processing, Post-Translational; RNA-Binding Proteins; Ubiquitination | 2023 |
The nutrient sensor OGT regulates Hipk stability and tumorigenic-like activities in
Topics: Acetylglucosamine; Animals; Carcinogenesis; Carrier Proteins; Cell Proliferation; Cells, Cultured; Drosophila melanogaster; Drosophila Proteins; Fibroblasts; Glucose; HEK293 Cells; Humans; MCF-7 Cells; Mice; N-Acetylglucosaminyltransferases; Phosphorylation; Protein Kinases; Protein Serine-Threonine Kinases; Protein Stability; Sweetening Agents | 2020 |
The essential role of YAP O-GlcNAcylation in high-glucose-stimulated liver tumorigenesis.
Topics: Acetylglucosamine; Adaptor Proteins, Signal Transducing; Animals; beta-Transducin Repeat-Containing Proteins; Carcinogenesis; Cell Line, Tumor; Gene Expression Regulation, Neoplastic; Glucose; Glycosylation; Humans; Liver Neoplasms; Male; Mice, Inbred BALB C; N-Acetylglucosaminyltransferases; Phosphoproteins; Protein Stability; Threonine; Transcription Factors; Transcription, Genetic; YAP-Signaling Proteins | 2017 |
Gastric gland mucin-specific O-glycan expression decreases with tumor progression from precursor lesions to pancreatic cancer.
Topics: Acetylglucosamine; Biomarkers, Tumor; Carbohydrate Conformation; Carcinogenesis; Carcinoma, Pancreatic Ductal; Disease Progression; Gastric Mucosa; Glycosylation; Humans; Mucin 5AC; Mucin-6; Pancreatic Neoplasms; Precancerous Conditions; Protein Processing, Post-Translational | 2017 |
Reciprocal Regulation Between O-GlcNAcylation and β-Catenin Facilitates Cell Viability and Inhibits Apoptosis in Liver Cancer.
Topics: Acetylglucosamine; Apoptosis; beta Catenin; Carcinogenesis; Cell Survival; Disease Progression; Hep G2 Cells; Humans; Liver Neoplasms; Nucleotidyltransferases; Up-Regulation | 2019 |
O-GlcNAcylation of ITGA5 facilitates the occurrence and development of colorectal cancer.
Topics: Acetylglucosamine; Acylation; Apoptosis; Carcinogenesis; Cell Line, Tumor; Cell Proliferation; Colorectal Neoplasms; Humans; Integrin alpha5; Up-Regulation | 2019 |